Amyloid-β (Aβ) immunotherapy induced microhemorrhages are linked to vascular inflammation and cerebrovascular damage in a mouse model of Alzheimer’s disease
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- Umfang
-
1 Online-Ressource.
- Sprache
-
Englisch
- Erschienen in
-
Amyloid-β (Aβ) immunotherapy induced microhemorrhages are linked to vascular inflammation and cerebrovascular damage in a mouse model of Alzheimer’s disease ; volume:19 ; number:1 ; day:21 ; month:10 ; year:2024 ; pages:1-16 ; date:12.2024
Molecular neurodegeneration ; 19, Heft 1 (21.10.2024), 1-16, 12.2024
- Urheber
-
Taylor, Xavier
Noristani, Harun N.
Fitzgerald, Griffin J.
Oluoch, Herold
Babb, Nick
McGathey, Tyler
Carter, Lindsay
Hole, Justin T.
Lacor, Pascale N.
DeMattos, Ronald B.
Wang, Yaming
- Beteiligte Personen und Organisationen
-
SpringerLink (Online service)
- DOI
-
10.1186/s13024-024-00758-0
- URN
-
urn:nbn:de:101:1-2501092112107.536457702207
- Rechteinformation
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
15.08.2025, 07:23 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Taylor, Xavier
- Noristani, Harun N.
- Fitzgerald, Griffin J.
- Oluoch, Herold
- Babb, Nick
- McGathey, Tyler
- Carter, Lindsay
- Hole, Justin T.
- Lacor, Pascale N.
- DeMattos, Ronald B.
- Wang, Yaming
- SpringerLink (Online service)
Ähnliche Objekte (12)
Nanoscale structure of amyloid-β plaques in Alzheimer’s disease
Medin co-aggregates with vascular amyloid-β in Alzheimer’s disease
Naringenin ameliorates amyloid-β pathology and neuroinflammation in Alzheimer’s disease
Blood amyloid-β oligomerization associated with neurodegeneration of Alzheimer’s disease
Clearance-Mechanismen von Amyloid-β bei der Alzheimer-Krankheit
Distinct amyloid-β and tau-associated microglia profiles in Alzheimer’s disease
Myelin dysfunction drives amyloid-β deposition in models of Alzheimer’s disease
Morphological and Molecular Profiling of Amyloid-β Species in Alzheimer’s Pathogenesis
Alzheimer’s disease neuropathological change three decades after iatrogenic amyloid-β transmission
Zinc ion rapidly induces toxic, off-pathway amyloid-β oligomers distinct from amyloid-β derived diffusible ligands in Alzheimer’s disease
A central role for dityrosine crosslinking of Amyloid-β in Alzheimer’s disease
Amyloid-β impairs mitochondrial dynamics and autophagy in Alzheimer’s disease experimental models
Nanoscale structure of amyloid-β plaques in Alzheimer’s disease
Medin co-aggregates with vascular amyloid-β in Alzheimer’s disease
Naringenin ameliorates amyloid-β pathology and neuroinflammation in Alzheimer’s disease
Blood amyloid-β oligomerization associated with neurodegeneration of Alzheimer’s disease
Clearance-Mechanismen von Amyloid-β bei der Alzheimer-Krankheit
Distinct amyloid-β and tau-associated microglia profiles in Alzheimer’s disease
Myelin dysfunction drives amyloid-β deposition in models of Alzheimer’s disease
Morphological and Molecular Profiling of Amyloid-β Species in Alzheimer’s Pathogenesis
Alzheimer’s disease neuropathological change three decades after iatrogenic amyloid-β transmission
Zinc ion rapidly induces toxic, off-pathway amyloid-β oligomers distinct from amyloid-β derived diffusible ligands in Alzheimer’s disease
A central role for dityrosine crosslinking of Amyloid-β in Alzheimer’s disease
Amyloid-β impairs mitochondrial dynamics and autophagy in Alzheimer’s disease experimental models
Nanoscale structure of amyloid-β plaques in Alzheimer’s disease
Medin co-aggregates with vascular amyloid-β in Alzheimer’s disease
Naringenin ameliorates amyloid-β pathology and neuroinflammation in Alzheimer’s disease
Blood amyloid-β oligomerization associated with neurodegeneration of Alzheimer’s disease
Clearance-Mechanismen von Amyloid-β bei der Alzheimer-Krankheit
Distinct amyloid-β and tau-associated microglia profiles in Alzheimer’s disease
Myelin dysfunction drives amyloid-β deposition in models of Alzheimer’s disease
Morphological and Molecular Profiling of Amyloid-β Species in Alzheimer’s Pathogenesis
Alzheimer’s disease neuropathological change three decades after iatrogenic amyloid-β transmission
Zinc ion rapidly induces toxic, off-pathway amyloid-β oligomers distinct from amyloid-β derived diffusible ligands in Alzheimer’s disease
A central role for dityrosine crosslinking of Amyloid-β in Alzheimer’s disease