SRC inhibition prevents P-cadherin mediated signaling and function in basal-like breast cancer cells

zu Verbundenen Objekten

Standort
Deutsche Nationalbibliothek Frankfurt am Main
ISSN
1478-811X
Umfang
Online-Ressource
Sprache
Englisch
Anmerkungen
online resource.

Erschienen in
SRC inhibition prevents P-cadherin mediated signaling and function in basal-like breast cancer cells ; volume:16 ; number:1 ; day:7 ; month:11 ; year:2018 ; pages:1-16 ; date:12.2018
Cell communication and signaling ; 16, Heft 1 (7.11.2018), 1-16, 12.2018

Urheber
Ribeiro, Ana Sofia
Nobre, Ana Rita
Mendes, Nuno
Almeida, João
Vieira, André Filipe
Sousa, Bárbara
Carvalho, Filomena A.
Monteiro, Joana
Polónia, António
Fonseca, Martina
Sanches, João Miguel
Santos, Nuno C.
Seruca, Raquel
Paredes, Joana
Beteiligte Personen und Organisationen
SpringerLink (Online service)

DOI
10.1186/s12964-018-0286-2
URN
urn:nbn:de:101:1-2018122522332541173112
Rechteinformation
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
Letzte Aktualisierung
14.08.2025, 10:49 MESZ

Datenpartner

Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Original beim Datenpartner anzeigen

Beteiligte

  • Ribeiro, Ana Sofia
  • Nobre, Ana Rita
  • Mendes, Nuno
  • Almeida, João
  • Vieira, André Filipe
  • Sousa, Bárbara
  • Carvalho, Filomena A.
  • Monteiro, Joana
  • Polónia, António
  • Fonseca, Martina
  • Sanches, João Miguel
  • Santos, Nuno C.
  • Seruca, Raquel
  • Paredes, Joana
  • SpringerLink (Online service)

Ähnliche Objekte (12)

The role of Met, Src and Stat3 in basal-like breast cancer invasion

The role of Met, Src and Stat3 in basal-like breast cancer invasion

Feline mammary basal-like adenocarcinomas: a potential model for human triple-negative breast cancer (TNBC) with basal-like subtype

Feline mammary basal-like adenocarcinomas: a potential model for human triple-negative breast cancer (TNBC) with basal-like subtype

Inactivation of LATS1/2 drives luminal-basal plasticity to initiate basal-like mammary carcinomas

Inactivation of LATS1/2 drives luminal-basal plasticity to initiate basal-like mammary carcinomas

Weight loss reduces basal-like breast cancer through kinome reprogramming

Weight loss reduces basal-like breast cancer through kinome reprogramming

Basal-like breast cancer: molecular profiles, clinical features and survival outcomes

Basal-like breast cancer: molecular profiles, clinical features and survival outcomes

Targeting BCL-2–expressing basal-like breast cancer with BH3-mimetics

Targeting BCL-2–expressing basal-like breast cancer with BH3-mimetics

The CD44+/CD24-phenotype is enriched in basal-like breast tumors

The CD44+/CD24-phenotype is enriched in basal-like breast tumors

Activation of Bivalent Gene POU4F1 Promotes and Maintains Basal‐like Breast Cancer

Activation of Bivalent Gene POU4F1 Promotes and Maintains Basal‐like Breast Cancer

Metabolic biomarkers for response to PI3K inhibition in basal-like breast cancer

Metabolic biomarkers for response to PI3K inhibition in basal-like breast cancer

Gene expression signatures of morphologically normal breast tissue identify basal-like tumors

Gene expression signatures of morphologically normal breast tissue identify basal-like tumors

ME1 promotes basal-like breast cancer progression and associates with poor prognosis

ME1 promotes basal-like breast cancer progression and associates with poor prognosis

Increased gene expression variability in BRCA1-associated and basal-like breast tumours

Increased gene expression variability in BRCA1-associated and basal-like breast tumours

Verbundene Objekte