Eight-fold increased COVID-19 mortality in autosomal dominant tubulointerstitial kidney disease due to MUC1 mutations: an observational study
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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1 Online-Ressource.
- Language
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Englisch
- Bibliographic citation
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Eight-fold increased COVID-19 mortality in autosomal dominant tubulointerstitial kidney disease due to MUC1 mutations: an observational study ; volume:25 ; number:1 ; day:18 ; month:12 ; year:2024 ; pages:1-10 ; date:12.2024
BMC nephrology ; 25, Heft 1 (18.12.2024), 1-10, 12.2024
- Creator
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Kidd, Kendrah O.
Williams, Adrienne H.
Taylor, Abbigail
Martin, Lauren
Robins, Victoria
Sayer, John A.
Olinger, Eric
Mabillard, Holly R.
Papagregoriou, Gregory
Deltas, Constantinos
Stavrou, Christoforos
Conlon, Peter J.
Hogan, Richard Edmund
Elhassan, Elhussein A.E
Springer, Drahomíra
Zima, Tomáš
Izzi, Claudia
Vrbacká, Alena
Piherová, Lenka
Pohludka, Michal
Radina, Martin
Vylet’al, Petr
Hodanova, Katerina
Zivna, Martina
Kmoch, Stanislav
Bleyer, Anthony J.
- Contributor
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SpringerLink (Online service)
- DOI
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10.1186/s12882-024-03896-1
- URN
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urn:nbn:de:101:1-2503010635375.259180682113
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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15.08.2025, 7:32 AM CEST
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Associated
- Kidd, Kendrah O.
- Williams, Adrienne H.
- Taylor, Abbigail
- Martin, Lauren
- Robins, Victoria
- Sayer, John A.
- Olinger, Eric
- Mabillard, Holly R.
- Papagregoriou, Gregory
- Deltas, Constantinos
- Stavrou, Christoforos
- Conlon, Peter J.
- Hogan, Richard Edmund
- Elhassan, Elhussein A.E
- Springer, Drahomíra
- Zima, Tomáš
- Izzi, Claudia
- Vrbacká, Alena
- Piherová, Lenka
- Pohludka, Michal
- Radina, Martin
- Vylet’al, Petr
- Hodanova, Katerina
- Zivna, Martina
- Kmoch, Stanislav
- Bleyer, Anthony J.
- SpringerLink (Online service)
Other Objects (12)
Plasma Mucin-1 (CA15-3) Levels in Autosomal Dominant Tubulointerstitial Kidney Disease due to MUC1 Mutations
Novel functional and genetic findings in autosomal dominant tubulointerstitial kidney disease
Allelic effects on uromodulin aggregates drive autosomal dominant tubulointerstitial kidney disease
Autosomal dominant tubulointerstitial kidney disease-UMOD is the most frequent non polycystic genetic kidney disease
MANF stimulates autophagy and restores mitochondrial homeostasis to treat autosomal dominant tubulointerstitial kidney disease in mice
Significance of kidney biopsy in autosomal dominant tubulointerstitial kidney disease-UMOD: is kidney biopsy truly nonspecific?
UMOD gene mutations in Chinese patients with autosomal dominant tubulointerstitial kidney disease: a pediatric case report and literature review
Recognizing IgG4-related tubulointerstitial nephritis
MUC1-Expression im Mammakarzinom und Korrelation mit der verändert glykosylierten Variante TA-MUC1
A novel likely pathogenic variant in the UMOD gene in a family with autosomal dominant tubulointerstitial kidney disease: a case report
Rekombinante Antikörper gegen tumorassoziiertes MUC1
Rekombinante Antikörper gegen tumorassoziiertes MUC1
Plasma Mucin-1 (CA15-3) Levels in Autosomal Dominant Tubulointerstitial Kidney Disease due to MUC1 Mutations
Novel functional and genetic findings in autosomal dominant tubulointerstitial kidney disease
Allelic effects on uromodulin aggregates drive autosomal dominant tubulointerstitial kidney disease
Autosomal dominant tubulointerstitial kidney disease-UMOD is the most frequent non polycystic genetic kidney disease
MANF stimulates autophagy and restores mitochondrial homeostasis to treat autosomal dominant tubulointerstitial kidney disease in mice
Significance of kidney biopsy in autosomal dominant tubulointerstitial kidney disease-UMOD: is kidney biopsy truly nonspecific?
UMOD gene mutations in Chinese patients with autosomal dominant tubulointerstitial kidney disease: a pediatric case report and literature review
Recognizing IgG4-related tubulointerstitial nephritis
MUC1-Expression im Mammakarzinom und Korrelation mit der verändert glykosylierten Variante TA-MUC1
A novel likely pathogenic variant in the UMOD gene in a family with autosomal dominant tubulointerstitial kidney disease: a case report
Rekombinante Antikörper gegen tumorassoziiertes MUC1
Rekombinante Antikörper gegen tumorassoziiertes MUC1
Plasma Mucin-1 (CA15-3) Levels in Autosomal Dominant Tubulointerstitial Kidney Disease due to MUC1 Mutations
Novel functional and genetic findings in autosomal dominant tubulointerstitial kidney disease
Allelic effects on uromodulin aggregates drive autosomal dominant tubulointerstitial kidney disease
Autosomal dominant tubulointerstitial kidney disease-UMOD is the most frequent non polycystic genetic kidney disease
MANF stimulates autophagy and restores mitochondrial homeostasis to treat autosomal dominant tubulointerstitial kidney disease in mice
Significance of kidney biopsy in autosomal dominant tubulointerstitial kidney disease-UMOD: is kidney biopsy truly nonspecific?
UMOD gene mutations in Chinese patients with autosomal dominant tubulointerstitial kidney disease: a pediatric case report and literature review
Recognizing IgG4-related tubulointerstitial nephritis
MUC1-Expression im Mammakarzinom und Korrelation mit der verändert glykosylierten Variante TA-MUC1
A novel likely pathogenic variant in the UMOD gene in a family with autosomal dominant tubulointerstitial kidney disease: a case report
Rekombinante Antikörper gegen tumorassoziiertes MUC1