Transcription stress at telomeres leads to cytosolic DNA release and paracrine senescence
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- Umfang
-
1 Online-Ressource.
- Sprache
-
Englisch
- Erschienen in
-
Transcription stress at telomeres leads to cytosolic DNA release and paracrine senescence ; volume:15 ; number:1 ; day:14 ; month:5 ; year:2024 ; pages:1-20 ; date:12.2024
Nature Communications ; 15, Heft 1 (14.5.2024), 1-20, 12.2024
- Urheber
-
Siametis, Athanasios
Stratigi, Kalliopi
Giamaki, Despoina
Chatzinikolaou, Georgia
Akalestou-Clocher, Alexia
Goulielmaki, Evi
Luke, Brian
Schumacher, Björn
Garinis, George A.
- Beteiligte Personen und Organisationen
-
SpringerLink (Online service)
- DOI
-
10.1038/s41467-024-48443-6
- URN
-
urn:nbn:de:101:1-2407240808075.786894360725
- Rechteinformation
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
14.08.2025, 10:52 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Siametis, Athanasios
- Stratigi, Kalliopi
- Giamaki, Despoina
- Chatzinikolaou, Georgia
- Akalestou-Clocher, Alexia
- Goulielmaki, Evi
- Luke, Brian
- Schumacher, Björn
- Garinis, George A.
- SpringerLink (Online service)
Ähnliche Objekte (12)
Telomeres, cardiovascular aging, and potential intervention for cellular senescence
Five dysfunctional telomeres predict onset of senescence in human cells
Npl3 stabilizes R-loops at telomeres to regulate replicative senescence
Telomeres, oxidative stress and inflammatory factors: partners in cellular senescence?
Paracrine cellular senescence exacerbates biliary injury and impairs regeneration
Npl3 stabilizes R-loops at telomeres to prevent accelerated replicative senescence
Npl3 stabilizes R‐loops at telomeres to prevent accelerated replicative senescence
Neutrophils induce paracrine telomere dysfunction and senescence in ROS‐dependent manner
Autocrine and paracrine effects of BRAF inhibitor induced senescence in melanoma
Human cytomegalovirus infection triggers a paracrine senescence loop in renal epithelial cells
Adamantinomatous craniopharyngioma as a model to understand paracrine and senescence-induced tumourigenesis
Retrotransposons and Telomeres
Telomeres, cardiovascular aging, and potential intervention for cellular senescence
Five dysfunctional telomeres predict onset of senescence in human cells
Npl3 stabilizes R-loops at telomeres to regulate replicative senescence
Telomeres, oxidative stress and inflammatory factors: partners in cellular senescence?
Paracrine cellular senescence exacerbates biliary injury and impairs regeneration
Npl3 stabilizes R-loops at telomeres to prevent accelerated replicative senescence
Npl3 stabilizes R‐loops at telomeres to prevent accelerated replicative senescence
Neutrophils induce paracrine telomere dysfunction and senescence in ROS‐dependent manner
Autocrine and paracrine effects of BRAF inhibitor induced senescence in melanoma
Human cytomegalovirus infection triggers a paracrine senescence loop in renal epithelial cells
Adamantinomatous craniopharyngioma as a model to understand paracrine and senescence-induced tumourigenesis
Retrotransposons and Telomeres
Telomeres, cardiovascular aging, and potential intervention for cellular senescence
Five dysfunctional telomeres predict onset of senescence in human cells
Npl3 stabilizes R-loops at telomeres to regulate replicative senescence
Telomeres, oxidative stress and inflammatory factors: partners in cellular senescence?
Paracrine cellular senescence exacerbates biliary injury and impairs regeneration
Npl3 stabilizes R-loops at telomeres to prevent accelerated replicative senescence
Npl3 stabilizes R‐loops at telomeres to prevent accelerated replicative senescence
Neutrophils induce paracrine telomere dysfunction and senescence in ROS‐dependent manner
Autocrine and paracrine effects of BRAF inhibitor induced senescence in melanoma
Human cytomegalovirus infection triggers a paracrine senescence loop in renal epithelial cells
Adamantinomatous craniopharyngioma as a model to understand paracrine and senescence-induced tumourigenesis