TRESK channel contribution to nociceptive sensory neurons excitability: modulation by nerve injury

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 1744-8069

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: TRESK channel contribution to nociceptive sensory neurons excitability: modulation by nerve injury ; volume:7 ; number:1 ; day:28 ; month:4 ; year:2011 ; pages:1-17 ; date:12.2011
Molecular pain ; 7, Heft 1 (28.4.2011), 1-17, 12.2011

Creator: Tulleuda, Astrid
Cokic, Barbara
Callejo, Gerard
Saiani, Barbara
Serra, Jordi
Gasull, Xavier

Contributor: SpringerLink (Online service)

DOI: 10.1186/1744-8069-7-30

URN: urn:nbn:de:101:1-2021080920093062788394

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 2025-08-14T10:49:16+0200

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Tulleuda, Astrid
Cokic, Barbara
Callejo, Gerard
Saiani, Barbara
Serra, Jordi
Gasull, Xavier
SpringerLink (Online service)

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SMN deficiency causes pain hypersensitivity in a mild SMA mouse model through enhancing excitability of nociceptive dorsal root ganglion neurons

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SMN deficiency causes pain hypersensitivity in a mild SMA mouse model through enhancing excitability of nociceptive dorsal root ganglion neurons

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TRESK channel contribution to nociceptive sensory neurons excitability: modulation by nerve injury

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Other Objects (12)

Collagens modulate pro-nociceptive processes in primary sensory neurons

ClC-3 regulates the excitability of nociceptive neurons and is involved in inflammatory processes within the spinal sensory pathway

Corrigendum: ClC-3 regulates the excitability of nociceptive neurons and is involved in inflammatory processes within the spinal sensory pathway

Delta opioid receptors on nociceptive sensory neurons mediate peripheral endogenous analgesia in colitis

Peripheral opioid receptors and inhibition of sensory neuron excitability following nerve injury

Estrogen signaling in nociceptive neurons

Polysulfide evokes acute pain through the activation of nociceptive TRPA1 in mouse sensory neurons

Nociceptive neurons detect cytokines in arthritis

Excitability of Aβ sensory neurons is altered in an animal model of peripheral neuropathy

Cdyl Deficiency Brakes Neuronal Excitability and Nociception through Promoting Kcnb1 Transcription in Peripheral Sensory Neurons

Cdyl Deficiency Brakes Neuronal Excitability and Nociception through Promoting Kcnb1 Transcription in Peripheral Sensory Neurons

SMN deficiency causes pain hypersensitivity in a mild SMA mouse model through enhancing excitability of nociceptive dorsal root ganglion neurons

Collagens modulate pro-nociceptive processes in primary sensory neurons

ClC-3 regulates the excitability of nociceptive neurons and is involved in inflammatory processes within the spinal sensory pathway

Corrigendum: ClC-3 regulates the excitability of nociceptive neurons and is involved in inflammatory processes within the spinal sensory pathway

Delta opioid receptors on nociceptive sensory neurons mediate peripheral endogenous analgesia in colitis

Peripheral opioid receptors and inhibition of sensory neuron excitability following nerve injury

Estrogen signaling in nociceptive neurons

Polysulfide evokes acute pain through the activation of nociceptive TRPA1 in mouse sensory neurons

Nociceptive neurons detect cytokines in arthritis

Excitability of Aβ sensory neurons is altered in an animal model of peripheral neuropathy

Cdyl Deficiency Brakes Neuronal Excitability and Nociception through Promoting Kcnb1 Transcription in Peripheral Sensory Neurons

Cdyl Deficiency Brakes Neuronal Excitability and Nociception through Promoting Kcnb1 Transcription in Peripheral Sensory Neurons

SMN deficiency causes pain hypersensitivity in a mild SMA mouse model through enhancing excitability of nociceptive dorsal root ganglion neurons

Collagens modulate pro-nociceptive processes in primary sensory neurons

ClC-3 regulates the excitability of nociceptive neurons and is involved in inflammatory processes within the spinal sensory pathway

Corrigendum: ClC-3 regulates the excitability of nociceptive neurons and is involved in inflammatory processes within the spinal sensory pathway

Delta opioid receptors on nociceptive sensory neurons mediate peripheral endogenous analgesia in colitis

Peripheral opioid receptors and inhibition of sensory neuron excitability following nerve injury

Estrogen signaling in nociceptive neurons

Polysulfide evokes acute pain through the activation of nociceptive TRPA1 in mouse sensory neurons

Nociceptive neurons detect cytokines in arthritis

Excitability of Aβ sensory neurons is altered in an animal model of peripheral neuropathy

Cdyl Deficiency Brakes Neuronal Excitability and Nociception through Promoting Kcnb1 Transcription in Peripheral Sensory Neurons

Cdyl Deficiency Brakes Neuronal Excitability and Nociception through Promoting Kcnb1 Transcription in Peripheral Sensory Neurons

SMN deficiency causes pain hypersensitivity in a mild SMA mouse model through enhancing excitability of nociceptive dorsal root ganglion neurons

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