- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
2041-1723
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Sumoylation regulates FMRP-mediated dendritic spine elimination and maturation ; volume:9 ; number:1 ; day:22 ; month:2 ; year:2018 ; pages:1-17 ; date:12.2018
Nature Communications ; 9, Heft 1 (22.2.2018), 1-17, 12.2018
- Keyword
-
Gehirn
Zentralnervensystem
- Creator
-
Khayachi, Anouar
- Contributor
-
Gwizdek, Carole
Poupon, Gwénola
Alcor, Damien
Chafai, Magda
Cassé, Frédéric
Maurin, Thomas
Prieto, Marta
Folci, Alessandra
Graeve, Fabienne De
Castagnola, Sara
Gautier, Romain
Schorova, Lenka
Loriol, Céline
Pronot, Marie
Besse, Florence
Brau, Frédéric
Deval, Emmanuel
Bardoni, Barbara
Martin, Stéphane
SpringerLink (Online service)
- DOI
-
10.1038/s41467-018-03222-y
- URN
-
urn:nbn:de:1111-2018040815318
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:54 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Khayachi, Anouar
- Gwizdek, Carole
- Poupon, Gwénola
- Alcor, Damien
- Chafai, Magda
- Cassé, Frédéric
- Maurin, Thomas
- Prieto, Marta
- Folci, Alessandra
- Graeve, Fabienne De
- Castagnola, Sara
- Gautier, Romain
- Schorova, Lenka
- Loriol, Céline
- Pronot, Marie
- Besse, Florence
- Brau, Frédéric
- Deval, Emmanuel
- Bardoni, Barbara
- Martin, Stéphane
- SpringerLink (Online service)
Other Objects (12)
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VPS35-deficiency results in an impaired AMPA receptor trafficking and decreased dendritic spine maturation
Amyloid plaque formation precedes dendritic spine loss
LOTUS suppresses amyloid β-induced dendritic spine elimination through the blockade of amyloid β binding to PirB
The role of PRG1 at the dendritic spine
Increased spine PIP3 is sequestered from dendritic shafts
A model of activity-dependent changes in dendritic spine density and spine structure
Hotspots of dendritic spine turnover facilitate clustered spine addition and learning and memory
Development of techniques for single dendritic spine analysis: spinomics
An automated open-source system for dendritic spine tracing
Diacylglycerol kinase β promotes dendritic outgrowth and spine maturation in developing hippocampal neurons
Ketamine and selective activation of parvalbumin interneurons inhibit stress-induced dendritic spine elimination
Cyclase-associated protein (CAP) inhibits inverted formin 2 (INF2) to induce dendritic spine maturation
VPS35-deficiency results in an impaired AMPA receptor trafficking and decreased dendritic spine maturation
Amyloid plaque formation precedes dendritic spine loss
LOTUS suppresses amyloid β-induced dendritic spine elimination through the blockade of amyloid β binding to PirB
The role of PRG1 at the dendritic spine
Increased spine PIP3 is sequestered from dendritic shafts
A model of activity-dependent changes in dendritic spine density and spine structure
Hotspots of dendritic spine turnover facilitate clustered spine addition and learning and memory
Development of techniques for single dendritic spine analysis: spinomics
An automated open-source system for dendritic spine tracing
Diacylglycerol kinase β promotes dendritic outgrowth and spine maturation in developing hippocampal neurons
Ketamine and selective activation of parvalbumin interneurons inhibit stress-induced dendritic spine elimination
Cyclase-associated protein (CAP) inhibits inverted formin 2 (INF2) to induce dendritic spine maturation
VPS35-deficiency results in an impaired AMPA receptor trafficking and decreased dendritic spine maturation
Amyloid plaque formation precedes dendritic spine loss
LOTUS suppresses amyloid β-induced dendritic spine elimination through the blockade of amyloid β binding to PirB
The role of PRG1 at the dendritic spine
Increased spine PIP3 is sequestered from dendritic shafts
A model of activity-dependent changes in dendritic spine density and spine structure
Hotspots of dendritic spine turnover facilitate clustered spine addition and learning and memory
Development of techniques for single dendritic spine analysis: spinomics