The molecular mechanisms of complement evasion by asexual blood stages and infective sporozoites of Plasmodium falciparum

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Alternative title: Die molekularen Mechanismen der Komplementvermeidung durch asexuelle Blutstadien und infektiöse Sporozoiten von Plasmodium falciparum

Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: Online-Ressource

Language: Englisch

Notes: RWTH Aachen University, Dissertation, 2018

Keyword: Blood
Plasmodium falciparum
Malaria

Event: Veröffentlichung

(where): Aachen

(who): Universitätsbibliothek der RWTH Aachen

(when): 2018

Creator: Araujo Rosa, Thiago Ferreira de

Contributor: Pradel, Gabriele
Rink, Lothar

DOI: 10.18154/RWTH-2018-227116

URN: urn:nbn:de:101:1-2019031506221644141033

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:50 AM CEST

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Associated

Araujo Rosa, Thiago Ferreira de
Pradel, Gabriele
Rink, Lothar
Universitätsbibliothek der RWTH Aachen

Time of origin

2018

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The molecular mechanisms of complement evasion by asexual blood stages and infective sporozoites of Plasmodium falciparum

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Other Objects (12)

Immune Evasion by Plasmodium falciparum Parasites: Converting a Host Protection Mechanism for the Parasite’s Benefit

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Immune Evasion by Plasmodium falciparum Parasites: Converting a Host Protection Mechanism for the Parasite’s Benefit

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The impact of human complement on the clinical outcome of malaria infection

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The Coming-Out of Malaria Gametocytes [Review Article]

The Multiple Roles of LCCL Domain-Containing Proteins for Malaria Parasite Transmission

EXIT — Wirtszellaustritt intrazellulärer Pathogene

EXIT — Wirtszellaustritt intrazellulärer Pathogene

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Functional characterization of three putative serpentine receptor proteins SR10, SR12 and 7 helix 2 important for the proliferation of asexual and sexual blood stages of the human malaria parasite Plasmodium falciparum

The cytosolic glyoxalases of Plasmodium falciparum are dispensable during asexual blood-stage development

Establishment of a cell culture model for the investigation of desmosome formation upon desmoglein 2 loss and arrhythmogenic cardiomyopathy related desmoglein 2 missense mutations

Immune Evasion by Plasmodium falciparum Parasites: Converting a Host Protection Mechanism for the Parasite’s Benefit

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The impact of human complement on the clinical outcome of malaria infection

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Establishment of a cell culture model for the investigation of desmosome formation upon desmoglein 2 loss and arrhythmogenic cardiomyopathy related desmoglein 2 missense mutations

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