Pharmacological EZH2 inhibition combined with retinoic acid treatment promotes differentiation and apoptosis in rhabdomyosarcoma cells

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Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: 1 Online-Ressource.

Language: Englisch

Bibliographic citation: Pharmacological EZH2 inhibition combined with retinoic acid treatment promotes differentiation and apoptosis in rhabdomyosarcoma cells ; volume:15 ; number:1 ; day:19 ; month:10 ; year:2023 ; pages:1-16 ; date:12.2023
Clinical epigenetics ; 15, Heft 1 (19.10.2023), 1-16, 12.2023

Creator: O’Brien, Eleanor
Tse, Carmen
Tracy, Ian
Reddin, Ian
Selfe, Joanna
Gibson, Jane
Tapper, William
Pengelly, Reuben J.
Gao, Jinhui
Aladowicz, Ewa
Petts, Gemma
Thway, Khin
Popov, Sergey
Kelsey, Anna
Underwood, Timothy J.
Shipley, Janet
Walters, Zoë S.

Contributor: SpringerLink (Online service)

DOI: 10.1186/s13148-023-01583-w

URN: urn:nbn:de:101:1-2024020908060234659654

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:32 AM CEST

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Associated

O’Brien, Eleanor
Tse, Carmen
Tracy, Ian
Reddin, Ian
Selfe, Joanna
Gibson, Jane
Tapper, William
Pengelly, Reuben J.
Gao, Jinhui
Aladowicz, Ewa
Petts, Gemma
Thway, Khin
Popov, Sergey
Kelsey, Anna
Underwood, Timothy J.
Shipley, Janet
Walters, Zoë S.
SpringerLink (Online service)

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Differential cooperation of oncogenes with p53 and Bax to induce apoptosis in rhabdomyosarcoma

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Selective BH3 mimetics synergize with BET inhibition to induce mitochondrial apoptosis in rhabdomyosarcoma cells

Concomitant epigenetic targeting of LSD1 and HDAC synergistically induces mitochondrial apoptosis in rhabdomyosarcoma cells

Co-targeting MCL-1 and ERK1/2 kinase induces mitochondrial apoptosis in rhabdomyosarcoma cells

EZH2 inhibition sensitizes retinoic acid-driven senescence in synovial sarcoma

Lineage of origin in rhabdomyosarcoma informs pharmacological response

Pharmacological inhibition of EZH2 disrupts the female germline epigenome

Pharmacological inhibition of EZH2 as a promising differentiation therapy in embryonal RMS

9-cis retinoic acid inhibition of activation-induced apoptosis is mediated via regulation of Fas ligand and requires retinoic acid receptor and retinoic X receptor activation

Differential cooperation of oncogenes with p53 and Bax to induce apoptosis in rhabdomyosarcoma

Differential apoptosis gene expressions of rhabdomyosarcoma cells in response to enterovirus 71 infection

FGFR4 and EZH2 inhibitors synergistically induce hepatocellular carcinoma apoptosis via repressing YAP signaling

Long noncoding RNA GAS5 promotes bladder cancer cells apoptosis through inhibiting EZH2 transcription

Selective BH3 mimetics synergize with BET inhibition to induce mitochondrial apoptosis in rhabdomyosarcoma cells

Concomitant epigenetic targeting of LSD1 and HDAC synergistically induces mitochondrial apoptosis in rhabdomyosarcoma cells

Co-targeting MCL-1 and ERK1/2 kinase induces mitochondrial apoptosis in rhabdomyosarcoma cells

EZH2 inhibition sensitizes retinoic acid-driven senescence in synovial sarcoma

Lineage of origin in rhabdomyosarcoma informs pharmacological response

Pharmacological inhibition of EZH2 disrupts the female germline epigenome

Pharmacological inhibition of EZH2 as a promising differentiation therapy in embryonal RMS

9-cis retinoic acid inhibition of activation-induced apoptosis is mediated via regulation of Fas ligand and requires retinoic acid receptor and retinoic X receptor activation

Differential cooperation of oncogenes with p53 and Bax to induce apoptosis in rhabdomyosarcoma

Differential apoptosis gene expressions of rhabdomyosarcoma cells in response to enterovirus 71 infection

FGFR4 and EZH2 inhibitors synergistically induce hepatocellular carcinoma apoptosis via repressing YAP signaling

Long noncoding RNA GAS5 promotes bladder cancer cells apoptosis through inhibiting EZH2 transcription

Selective BH3 mimetics synergize with BET inhibition to induce mitochondrial apoptosis in rhabdomyosarcoma cells

Concomitant epigenetic targeting of LSD1 and HDAC synergistically induces mitochondrial apoptosis in rhabdomyosarcoma cells

Co-targeting MCL-1 and ERK1/2 kinase induces mitochondrial apoptosis in rhabdomyosarcoma cells

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Pharmacological EZH2 inhibition combined with retinoic acid treatment promotes differentiation and apoptosis in rhabdomyosarcoma cells

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Other Objects (12)

EZH2 inhibition sensitizes retinoic acid-driven senescence in synovial sarcoma

Lineage of origin in rhabdomyosarcoma informs pharmacological response

Pharmacological inhibition of EZH2 disrupts the female germline epigenome

Pharmacological inhibition of EZH2 as a promising differentiation therapy in embryonal RMS

9-cis retinoic acid inhibition of activation-induced apoptosis is mediated via regulation of Fas ligand and requires retinoic acid receptor and retinoic X receptor activation

Differential cooperation of oncogenes with p53 and Bax to induce apoptosis in rhabdomyosarcoma

Differential apoptosis gene expressions of rhabdomyosarcoma cells in response to enterovirus 71 infection

FGFR4 and EZH2 inhibitors synergistically induce hepatocellular carcinoma apoptosis via repressing YAP signaling

Long noncoding RNA GAS5 promotes bladder cancer cells apoptosis through inhibiting EZH2 transcription

Selective BH3 mimetics synergize with BET inhibition to induce mitochondrial apoptosis in rhabdomyosarcoma cells

Concomitant epigenetic targeting of LSD1 and HDAC synergistically induces mitochondrial apoptosis in rhabdomyosarcoma cells

Co-targeting MCL-1 and ERK1/2 kinase induces mitochondrial apoptosis in rhabdomyosarcoma cells

EZH2 inhibition sensitizes retinoic acid-driven senescence in synovial sarcoma

Lineage of origin in rhabdomyosarcoma informs pharmacological response

Pharmacological inhibition of EZH2 disrupts the female germline epigenome

Pharmacological inhibition of EZH2 as a promising differentiation therapy in embryonal RMS

9-cis retinoic acid inhibition of activation-induced apoptosis is mediated via regulation of Fas ligand and requires retinoic acid receptor and retinoic X receptor activation

Differential cooperation of oncogenes with p53 and Bax to induce apoptosis in rhabdomyosarcoma

Differential apoptosis gene expressions of rhabdomyosarcoma cells in response to enterovirus 71 infection

FGFR4 and EZH2 inhibitors synergistically induce hepatocellular carcinoma apoptosis via repressing YAP signaling

Long noncoding RNA GAS5 promotes bladder cancer cells apoptosis through inhibiting EZH2 transcription

Selective BH3 mimetics synergize with BET inhibition to induce mitochondrial apoptosis in rhabdomyosarcoma cells

Concomitant epigenetic targeting of LSD1 and HDAC synergistically induces mitochondrial apoptosis in rhabdomyosarcoma cells

Co-targeting MCL-1 and ERK1/2 kinase induces mitochondrial apoptosis in rhabdomyosarcoma cells

EZH2 inhibition sensitizes retinoic acid-driven senescence in synovial sarcoma

Lineage of origin in rhabdomyosarcoma informs pharmacological response

Pharmacological inhibition of EZH2 disrupts the female germline epigenome

Pharmacological inhibition of EZH2 as a promising differentiation therapy in embryonal RMS

9-cis retinoic acid inhibition of activation-induced apoptosis is mediated via regulation of Fas ligand and requires retinoic acid receptor and retinoic X receptor activation

Differential cooperation of oncogenes with p53 and Bax to induce apoptosis in rhabdomyosarcoma

Differential apoptosis gene expressions of rhabdomyosarcoma cells in response to enterovirus 71 infection

FGFR4 and EZH2 inhibitors synergistically induce hepatocellular carcinoma apoptosis via repressing YAP signaling

Long noncoding RNA GAS5 promotes bladder cancer cells apoptosis through inhibiting EZH2 transcription

Selective BH3 mimetics synergize with BET inhibition to induce mitochondrial apoptosis in rhabdomyosarcoma cells

Concomitant epigenetic targeting of LSD1 and HDAC synergistically induces mitochondrial apoptosis in rhabdomyosarcoma cells

Co-targeting MCL-1 and ERK1/2 kinase induces mitochondrial apoptosis in rhabdomyosarcoma cells

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