Loss of heterozygosity at thymidylate synthase locus in Barrett's metaplasia, dysplasia, and carcinoma sequences
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1471-2407
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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Loss of heterozygosity at thymidylate synthase locus in Barrett's metaplasia, dysplasia, and carcinoma sequences ; volume:9 ; number:1 ; day:21 ; month:5 ; year:2009 ; pages:1-9 ; date:12.2009
BMC cancer ; 9, Heft 1 (21.5.2009), 1-9, 12.2009
- Creator
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Kuramochi, Hidekazu
- Contributor
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Uchida, Kazumi
Peters, Jeffery H.
Shimizu, Daisuke
Vallbohmer, Daniel
Schneider, Sylke
Danenberg, Kathleen D.
Danenberg, Peter V.
SpringerLink (Online service)
- DOI
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10.1186/1471-2407-9-157
- URN
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urn:nbn:de:1111-2016030350978
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
03.03.2128, 12:00 AM CET
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Kuramochi, Hidekazu
- Uchida, Kazumi
- Peters, Jeffery H.
- Shimizu, Daisuke
- Vallbohmer, Daniel
- Schneider, Sylke
- Danenberg, Kathleen D.
- Danenberg, Peter V.
- SpringerLink (Online service)
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Low-grade dysplasia on Barrett’s esophagus: visible or not?
Targeted Hsp70 fluorescence molecular endoscopy detects dysplasia in Barrett’s esophagus
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Length of Barrett’s esophagus in the presence of low-grade dysplasia, high-grade dysplasia, and adenocarcinoma
Diffuse endoscopically visible, predominantly low grade dysplasia in Barrett’s esophagus (with video)
Exploring the incidence of dysplasia or adenocarcinoma in early onset Barrett’s esophagus
Correlation of the presence and extent of loss of heterozygosity mutations with histological classifications of Barrett’s esophagus
Regulation of metaplasia and dysplasia in the stomach by the stromal microenvironment
Recurrent intestinal metaplasia at the gastroesophageal junction following endoscopic eradication of dysplastic Barrett’s esophagus may not be benign
Notch signaling drives development of Barrett’s metaplasia from Dclk1-positive epithelial tuft cells in the murine gastric mucosa
Symptomatic esophageal stricture and buried metaplasia after radiofrequency ablation of Barrett’s esophagus
Adenocarcinoma of the cervical esophagus arising within a long segment of Barrett’s metaplasia
Low-grade dysplasia on Barrett’s esophagus: visible or not?
Targeted Hsp70 fluorescence molecular endoscopy detects dysplasia in Barrett’s esophagus
Targeted Hsp70 fluorescence molecular endoscopy detects dysplasia in Barrett’s esophagus
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Diffuse endoscopically visible, predominantly low grade dysplasia in Barrett’s esophagus (with video)
Exploring the incidence of dysplasia or adenocarcinoma in early onset Barrett’s esophagus
Correlation of the presence and extent of loss of heterozygosity mutations with histological classifications of Barrett’s esophagus
Regulation of metaplasia and dysplasia in the stomach by the stromal microenvironment
Recurrent intestinal metaplasia at the gastroesophageal junction following endoscopic eradication of dysplastic Barrett’s esophagus may not be benign