Activation of hepatic stellate cells by the ubiquitin C-terminal hydrolase 1 protein secreted from hepatitis C virus-infected hepatocytes
- Standort
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2045-2322
- Umfang
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Online-Ressource
- Sprache
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Englisch
- Anmerkungen
-
online resource.
- Erschienen in
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Activation of hepatic stellate cells by the ubiquitin C-terminal hydrolase 1 protein secreted from hepatitis C virus-infected hepatocytes ; volume:7 ; number:1 ; day:30 ; month:6 ; year:2017 ; pages:1-12 ; date:12.2017
Scientific reports ; 7, Heft 1 (30.6.2017), 1-12, 12.2017
- Urheber
-
Cheng, Ju-Chien
Tseng, Ching-Ping
Liao, Mei-Huei
Peng, Cheng-Yuan
Yu, Jau-Song
Chuang, Po-Heng
Huang, Jing-Tang
Chen, Jeremy J. W.
- Beteiligte Personen und Organisationen
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SpringerLink (Online service)
- DOI
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10.1038/s41598-017-04259-7
- URN
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urn:nbn:de:101:1-2019102322352040192263
- Rechteinformation
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
15.08.2025, 07:21 MESZ
Datenpartner
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Beteiligte
- Cheng, Ju-Chien
- Tseng, Ching-Ping
- Liao, Mei-Huei
- Peng, Cheng-Yuan
- Yu, Jau-Song
- Chuang, Po-Heng
- Huang, Jing-Tang
- Chen, Jeremy J. W.
- SpringerLink (Online service)
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Generation of proliferating hepatocytes, liver sinusoidal endothelial cells and stellate cells and establishment of a genotoxicity assay based on proliferating hepatocytes
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Disruption of Trichoderma reesei cre2, encoding an ubiquitin C-terminal hydrolase, results in increased cellulase activity
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Drosophila Ubiquitin C-Terminal Hydrolase Knockdown Model of Parkinson’s Disease
Evaluation of ubiquitin C-terminal hydrolase-L1 enzyme levels in patients with epilepsy
Extracellular vesicles from steatotic hepatocytes provoke pro-fibrotic responses in cultured stellate cells
Retinol from hepatic stellate cells via STRA6 induces lipogenesis on hepatocytes during fibrosis
Hepatocyte-derived exosomal miR-146a-5p inhibits hepatic stellate cell EMT process: a crosstalk between hepatocytes and hepatic stellate cells
Generation of proliferating hepatocytes, liver sinusoidal endothelial cells and stellate cells and establishment of a genotoxicity assay based on proliferating hepatocytes
Generation of proliferating hepatocytes, liver sinusoidal endothelial cells and stellate cells and establishment of a genotoxicity assay based on proliferating hepatocytes
Ubiquitin C-terminal hydrolase-L1 (UCH-L1) stabilizes NOXA and impacts on cancer chemosusceptibility
Ubiquitin C-terminal hydrolase isozyme L1 is associated with shelterin complex at interstitial telomeric sites
Pathological Microenvironment‐Remodeling Nanoparticles to Alleviate Liver Fibrosis: Reversing Hepatocytes‐Hepatic Stellate Cells Malignant Crosstalk
Disruption of Trichoderma reesei cre2, encoding an ubiquitin C-terminal hydrolase, results in increased cellulase activity
GLP-1 and GIP agonism has no direct actions in human hepatocytes or hepatic stellate cells
Drosophila Ubiquitin C-Terminal Hydrolase Knockdown Model of Parkinson’s Disease
Evaluation of ubiquitin C-terminal hydrolase-L1 enzyme levels in patients with epilepsy
Extracellular vesicles from steatotic hepatocytes provoke pro-fibrotic responses in cultured stellate cells
Retinol from hepatic stellate cells via STRA6 induces lipogenesis on hepatocytes during fibrosis
Hepatocyte-derived exosomal miR-146a-5p inhibits hepatic stellate cell EMT process: a crosstalk between hepatocytes and hepatic stellate cells
Generation of proliferating hepatocytes, liver sinusoidal endothelial cells and stellate cells and establishment of a genotoxicity assay based on proliferating hepatocytes
Generation of proliferating hepatocytes, liver sinusoidal endothelial cells and stellate cells and establishment of a genotoxicity assay based on proliferating hepatocytes
Ubiquitin C-terminal hydrolase-L1 (UCH-L1) stabilizes NOXA and impacts on cancer chemosusceptibility
Ubiquitin C-terminal hydrolase isozyme L1 is associated with shelterin complex at interstitial telomeric sites
Pathological Microenvironment‐Remodeling Nanoparticles to Alleviate Liver Fibrosis: Reversing Hepatocytes‐Hepatic Stellate Cells Malignant Crosstalk
Disruption of Trichoderma reesei cre2, encoding an ubiquitin C-terminal hydrolase, results in increased cellulase activity