Hochschulschrift
Protein moonlighting : myeloid cell-derived BRICK1 enhances tissue repair after acute myocardial infarction
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Dimensions
-
30 cm
- Extent
-
114 Seiten
- Language
-
Englisch
- Notes
-
Illustrationen, Diagramme
Medizinische Hochschule Hannover, Dissertation, 2021
- Event
-
Veröffentlichung
- (where)
-
Hannover
- (when)
-
November 2020
- Creator
- Contributor
-
Wollert, Kai Christoph
Kraft, Theresia
Thum, Thomas
Medizinische Hochschule Hannover
Hannover Biomedical Research School
Institut für Molekulare und Translationale Therapiestrategien
Klinik für Kardiologie und Angiologie
- Table of contents
- Rights
-
Bei diesem Objekt liegt nur das Inhaltsverzeichnis digital vor. Der Zugriff darauf ist unbeschränkt möglich.
- Last update
-
13.03.2025, 5:50 AM CET
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Object type
- Hochschulschrift
Associated
- Sandu, Mircea-Andrei
- Wollert, Kai Christoph
- Kraft, Theresia
- Thum, Thomas
- Medizinische Hochschule Hannover
- Hannover Biomedical Research School
- Institut für Molekulare und Translationale Therapiestrategien
- Klinik für Kardiologie und Angiologie
Time of origin
- November 2020
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Myocardial infarction and psychosocial risks
Tissue factor cytoplasmic tail regulates myeloid cell activation and superoxide formation in chronic myocardial infarction
Syncope after myocardial infarction : electrophysiologic findings
Prognosis after ST-elevation myocardial infarction
Psychological problems before and after myocardial infarction
Respiration and risk stratification after myocardial infarction
5-Lipoxygenase facilitates healing after myocardial infarction
Adrenergic system and ventricular arrhythmias in myocardial infarction
Autonomic function after myocardial infarction : an epidemiological approach
Increased erythropoietin production after myocardial infarction in mice