Early life sleep disruption potentiates lasting sex-specific changes in behavior in genetically vulnerable Shank3 heterozygous autism model mice
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2040-2392
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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Early life sleep disruption potentiates lasting sex-specific changes in behavior in genetically vulnerable Shank3 heterozygous autism model mice ; volume:13 ; number:1 ; day:29 ; month:8 ; year:2022 ; pages:1-19 ; date:12.2022
Molecular autism ; 13, Heft 1 (29.8.2022), 1-19, 12.2022
- Creator
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Lord, Julia S.
Gay, Sean M.
Harper, Kathryn M.
Nikolova, Viktoriya D.
Smith, Kirsten M.
Moy, Sheryl S.
Diering, Graham H.
- Contributor
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SpringerLink (Online service)
- DOI
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10.1186/s13229-022-00514-5
- URN
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urn:nbn:de:101:1-2022111621062505074139
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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15.08.2025, 7:31 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Lord, Julia S.
- Gay, Sean M.
- Harper, Kathryn M.
- Nikolova, Viktoriya D.
- Smith, Kirsten M.
- Moy, Sheryl S.
- Diering, Graham H.
- SpringerLink (Online service)
Other Objects (12)
Transcriptional and functional complexity of Shank3 provides a molecular framework to understand the phenotypic heterogeneity of SHANK3 causing autism and Shank3 mutant mice
Pre-implantation alcohol exposure induces lasting sex-specific DNA methylation programming errors in the developing forebrain
SHANK3 deficiency leads to myelin dysregulation in a Shank3∆11(-/-) mouse model and human iPSC-derived cerebral organoids
Funktionelle Analyse von SHANK3-Mutationen assoziiert mit Synaptopathien
Microglia unravel immune-associated alterations in Shank3 deficiency
New insights into SHANK3-associated diseases and treatment options
A lasting peace
Wittgenstein's lasting significance
Single-molecule fluorescence in-situ hybridization reveals that human SHANK3 mRNA expression varies during development and in autism-associated SHANK3 heterozygosity
ProSAP2/Shank3 deficiency abrogates synaptic plasticity after brain trauma
Haploinsufficiency of Shank3 increases the orientation selectivity of V1 neurons
Expanding the Nude SCID/CID Phenotype Associated with FOXN1 Homozygous, Compound Heterozygous, or Heterozygous Mutations
Transcriptional and functional complexity of Shank3 provides a molecular framework to understand the phenotypic heterogeneity of SHANK3 causing autism and Shank3 mutant mice
Pre-implantation alcohol exposure induces lasting sex-specific DNA methylation programming errors in the developing forebrain
SHANK3 deficiency leads to myelin dysregulation in a Shank3∆11(-/-) mouse model and human iPSC-derived cerebral organoids
Funktionelle Analyse von SHANK3-Mutationen assoziiert mit Synaptopathien
Microglia unravel immune-associated alterations in Shank3 deficiency
New insights into SHANK3-associated diseases and treatment options
A lasting peace
Wittgenstein's lasting significance
Single-molecule fluorescence in-situ hybridization reveals that human SHANK3 mRNA expression varies during development and in autism-associated SHANK3 heterozygosity
ProSAP2/Shank3 deficiency abrogates synaptic plasticity after brain trauma
Haploinsufficiency of Shank3 increases the orientation selectivity of V1 neurons
Expanding the Nude SCID/CID Phenotype Associated with FOXN1 Homozygous, Compound Heterozygous, or Heterozygous Mutations
Transcriptional and functional complexity of Shank3 provides a molecular framework to understand the phenotypic heterogeneity of SHANK3 causing autism and Shank3 mutant mice
Pre-implantation alcohol exposure induces lasting sex-specific DNA methylation programming errors in the developing forebrain
SHANK3 deficiency leads to myelin dysregulation in a Shank3∆11(-/-) mouse model and human iPSC-derived cerebral organoids
Funktionelle Analyse von SHANK3-Mutationen assoziiert mit Synaptopathien
Microglia unravel immune-associated alterations in Shank3 deficiency
New insights into SHANK3-associated diseases and treatment options
A lasting peace
Wittgenstein's lasting significance
Single-molecule fluorescence in-situ hybridization reveals that human SHANK3 mRNA expression varies during development and in autism-associated SHANK3 heterozygosity
ProSAP2/Shank3 deficiency abrogates synaptic plasticity after brain trauma
Haploinsufficiency of Shank3 increases the orientation selectivity of V1 neurons