Three-dimensional human pluripotent stem cell-derived cardiomyocyte models for investigating cardiac hypertrophy
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
Hannover, Medizinische Hochschule Hannover, Dissertation, 2023
- Keyword
-
Herzhypertrophie
Tiermodell
Signaltransduktion
Herzmuskelzelle
Herzinsuffizienz
- Event
-
Veröffentlichung
- (where)
-
Hannover
- (who)
-
Bibliothek der Medizinischen Hochschule Hannover
- (when)
-
2024
- Creator
- Contributor
-
Zweigerdt, Robert
Kraft, Theresia
Kalies, Stefan Michael Klaus
Hannover Biomedical Research School
Klinik für Allgemein-, Viszeral- und Transplantationschirurgie
Leibniz-Forschungslaboratorien für Biotechnologie und Künstliche Organe
- DOI
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10.26068/mhhrpm/20240219-001
- URN
-
urn:nbn:de:101:1-2024030809204688686830
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
25.03.2025, 1:50 PM CET
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Biswanath Devadas, Santoshi
- Zweigerdt, Robert
- Kraft, Theresia
- Kalies, Stefan Michael Klaus
- Hannover Biomedical Research School
- Klinik für Allgemein-, Viszeral- und Transplantationschirurgie
- Leibniz-Forschungslaboratorien für Biotechnologie und Künstliche Organe
- Bibliothek der Medizinischen Hochschule Hannover
Time of origin
- 2024
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Electrophysiological characterization of embryonic and induced pluripotent stem cell-derived cardiomyocytes
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Inhibition of cardiomyocyte hypertrophy by heterocyclic thiourylenes in vitro
Characterization of pluripotent stem cell-derived cardiomyocytes
Sumoylation-independent activation of Calcineurin-NFAT-signaling via SUMO2 mediates cardiomyocyte hypertrophy
Sumoylation-independent activation of calcineurin-NFAT-signaling via SUMO2 causes cardiomyocyte hypertrophy
Cardiomyocyte hyperplasia and immaturity but not hypertrophy are characteristic features of patients with RASopathies
Altered cellular protein quality control system modulates cardiomyocyte function in volume overload-induced hypertrophy
Amlexanox and forskolin prevents isoproterenol-induced cardiomyopathy by subduing cardiomyocyte hypertrophy and maladaptive inflammatory responses
Angiotensin-(1-9) prevents cardiomyocyte hypertrophy via miR-129-3p/PKIA/PKA signaling pathway
Improving connexin 43 expression in induced pluripotent stem cell-derived cardiomyocytes
Electrophysiological characterization of embryonic and induced pluripotent stem cell-derived cardiomyocytes
Contribution of human stem cell-derived cardiomyocyte subtypes to bioartificial cardiac tissue formation and function
Characterization of induced pluripotent stem cell-derived cardiomyocytes under different maturation media
Inhibition of cardiomyocyte hypertrophy by heterocyclic thiourylenes in vitro
Characterization of pluripotent stem cell-derived cardiomyocytes
Sumoylation-independent activation of Calcineurin-NFAT-signaling via SUMO2 mediates cardiomyocyte hypertrophy
Sumoylation-independent activation of calcineurin-NFAT-signaling via SUMO2 causes cardiomyocyte hypertrophy
Cardiomyocyte hyperplasia and immaturity but not hypertrophy are characteristic features of patients with RASopathies
Altered cellular protein quality control system modulates cardiomyocyte function in volume overload-induced hypertrophy
Amlexanox and forskolin prevents isoproterenol-induced cardiomyopathy by subduing cardiomyocyte hypertrophy and maladaptive inflammatory responses
Angiotensin-(1-9) prevents cardiomyocyte hypertrophy via miR-129-3p/PKIA/PKA signaling pathway
Improving connexin 43 expression in induced pluripotent stem cell-derived cardiomyocytes
Electrophysiological characterization of embryonic and induced pluripotent stem cell-derived cardiomyocytes
Contribution of human stem cell-derived cardiomyocyte subtypes to bioartificial cardiac tissue formation and function