Preclinical studies of Apogossypolone: a new nonpeptidic pan small-molecule inhibitor of Bcl-2, Bcl-XL and Mcl-1 proteins in Follicular Small Cleaved Cell Lymphoma model
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1476-4598
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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Preclinical studies of Apogossypolone: a new nonpeptidic pan small-molecule inhibitor of Bcl-2, Bcl-XL and Mcl-1 proteins in Follicular Small Cleaved Cell Lymphoma model ; volume:7 ; number:1 ; day:14 ; month:2 ; year:2008 ; pages:1-10 ; date:12.2008
Molecular cancer ; 7, Heft 1 (14.2.2008), 1-10, 12.2008
- Creator
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Arnold, Alan A.
Aboukameel, Amro
Chen, Jianyong
Yang, Dajun
Wang, Shaomeng
Al-Katib, Ayad
Mohammad, Ramzi M.
- Contributor
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SpringerLink (Online service)
- DOI
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10.1186/1476-4598-7-20
- URN
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urn:nbn:de:101:1-2021082319445052709016
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:57 AM CEST
Data provider
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Associated
- Arnold, Alan A.
- Aboukameel, Amro
- Chen, Jianyong
- Yang, Dajun
- Wang, Shaomeng
- Al-Katib, Ayad
- Mohammad, Ramzi M.
- SpringerLink (Online service)
Other Objects (12)
Rolle von Bcl-2, Bcl-xl und Mcl-1 für die Apoptoseresistenz und Chemoresistenz des Pankreaskarzinoms
BCL-XL and MCL-1 are the key BCL-2 family proteins in melanoma cell survival
Targeting antiapoptotic proteins Bcl-xL and Mcl-1 in cholangiocarcinoma
Glutathione and Bcl-2 targeting facilitates elimination by chemoradiotherapy of human A375 melanoma xenografts overexpressing bcl-xl, bcl-2, and mcl-1
A direct comparison of selective BH3-mimetics reveals BCL-XL, BCL-2 and MCL-1 as promising therapeutic targets in neuroblastoma
The downregulation of Mcl-1 via USP9X inhibition sensitizes solid tumors to Bcl-xl inhibition
Bcl-XL but Not Bcl-2 is a potential target in medulloblastoma therapy
Regulation of Bcl-xL activity in apoptosis
Bcl-xl as the most promising Bcl-2 family member in targeted treatment of chondrosarcoma
BH4 domain peptides derived from Bcl-2/Bcl-XL as novel tools against acute pancreatitis
Dual inhibition of anti-apoptotic proteins BCL-XL and MCL-1 enhances cytotoxicity of Nasopharyngeal carcinoma cells
Photodynamic therapy combined with Bcl-2/Bcl-xL inhibition increases the Noxa/Mcl-1 ratio independent of usp9X and synergistically enhances apoptosis in glioblastoma
Rolle von Bcl-2, Bcl-xl und Mcl-1 für die Apoptoseresistenz und Chemoresistenz des Pankreaskarzinoms
BCL-XL and MCL-1 are the key BCL-2 family proteins in melanoma cell survival
Targeting antiapoptotic proteins Bcl-xL and Mcl-1 in cholangiocarcinoma
Glutathione and Bcl-2 targeting facilitates elimination by chemoradiotherapy of human A375 melanoma xenografts overexpressing bcl-xl, bcl-2, and mcl-1
A direct comparison of selective BH3-mimetics reveals BCL-XL, BCL-2 and MCL-1 as promising therapeutic targets in neuroblastoma
The downregulation of Mcl-1 via USP9X inhibition sensitizes solid tumors to Bcl-xl inhibition
Bcl-XL but Not Bcl-2 is a potential target in medulloblastoma therapy
Regulation of Bcl-xL activity in apoptosis
Bcl-xl as the most promising Bcl-2 family member in targeted treatment of chondrosarcoma
BH4 domain peptides derived from Bcl-2/Bcl-XL as novel tools against acute pancreatitis
Dual inhibition of anti-apoptotic proteins BCL-XL and MCL-1 enhances cytotoxicity of Nasopharyngeal carcinoma cells
Photodynamic therapy combined with Bcl-2/Bcl-xL inhibition increases the Noxa/Mcl-1 ratio independent of usp9X and synergistically enhances apoptosis in glioblastoma
Rolle von Bcl-2, Bcl-xl und Mcl-1 für die Apoptoseresistenz und Chemoresistenz des Pankreaskarzinoms
BCL-XL and MCL-1 are the key BCL-2 family proteins in melanoma cell survival
Targeting antiapoptotic proteins Bcl-xL and Mcl-1 in cholangiocarcinoma
Glutathione and Bcl-2 targeting facilitates elimination by chemoradiotherapy of human A375 melanoma xenografts overexpressing bcl-xl, bcl-2, and mcl-1
A direct comparison of selective BH3-mimetics reveals BCL-XL, BCL-2 and MCL-1 as promising therapeutic targets in neuroblastoma
The downregulation of Mcl-1 via USP9X inhibition sensitizes solid tumors to Bcl-xl inhibition
Bcl-XL but Not Bcl-2 is a potential target in medulloblastoma therapy
Regulation of Bcl-xL activity in apoptosis
Bcl-xl as the most promising Bcl-2 family member in targeted treatment of chondrosarcoma
BH4 domain peptides derived from Bcl-2/Bcl-XL as novel tools against acute pancreatitis
Dual inhibition of anti-apoptotic proteins BCL-XL and MCL-1 enhances cytotoxicity of Nasopharyngeal carcinoma cells