Stress induced lncRNA Lockd inhibits proliferation and angiogenesis of myocardial endothelial cells and promotes maladaptive cardiac remodelling during pressure overload
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
Heidelberg, Universität Heidelberg, Dissertation, 2023
- Keyword
-
Angiogenese
Tiermodell
Endothelzelle
Herzmuskelzelle
Herzhypertrophie
- Event
-
Veröffentlichung
- (where)
-
Heidelberg
- (who)
-
Universitätsbibliothek Heidelberg
- (when)
-
2023
- Creator
- Contributor
-
Wieland, Thomas
- DOI
-
10.11588/heidok.00034025
- URN
-
urn:nbn:de:bsz:16-heidok-340251
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:27 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Grein, Steve
- Wieland, Thomas
- Universitätsbibliothek Heidelberg
Time of origin
- 2023
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Maladaptive response following glucose overload in GLUT4-overexpressing H9C2 cardiomyoblasts
The role of Cullin-RING E3 ubiquitin ligase 7 in maladaptive cardiac remodelling
Effects of Multikinase inhibitors on pressure overload-induced right ventricular remodelling
FHL-1 is not involved in pressure overload-induced maladaptive right ventricular remodeling and dysfunction
Loss of MD1 exacerbates pressure overload-induced left ventricular structural and electrical remodelling
Hydrostatic pressure drives sprouting angiogenesis via adherens junction remodelling and YAP signalling
Early dysregulation of cardiac-specific microRNA-208a is linked to maladaptive cardiac remodelling in diabetic myocardium
Peptidylarginine deiminase type IV sustains inflammatory response and drives maladaptive cardiac remodelling after myocardial infarction in mice
Exosomal lncRNA SNHG12 promotes angiogenesis and breast cancer progression
Characterization of biventricular alterations in myocardial (reverse) remodelling in aortic banding-induced chronic pressure overload
Montelukast, an available and safe anti-asthmatic drug, prevents maladaptive remodelling and maintains cardiac functionality following myocardial infarction
VEGF ‐ A regulated by progesterone governs uterine angiogenesis and vascular remodelling during pregnancy
Maladaptive response following glucose overload in GLUT4-overexpressing H9C2 cardiomyoblasts
The role of Cullin-RING E3 ubiquitin ligase 7 in maladaptive cardiac remodelling
Effects of Multikinase inhibitors on pressure overload-induced right ventricular remodelling
FHL-1 is not involved in pressure overload-induced maladaptive right ventricular remodeling and dysfunction
Loss of MD1 exacerbates pressure overload-induced left ventricular structural and electrical remodelling
Hydrostatic pressure drives sprouting angiogenesis via adherens junction remodelling and YAP signalling
Early dysregulation of cardiac-specific microRNA-208a is linked to maladaptive cardiac remodelling in diabetic myocardium
Peptidylarginine deiminase type IV sustains inflammatory response and drives maladaptive cardiac remodelling after myocardial infarction in mice
Exosomal lncRNA SNHG12 promotes angiogenesis and breast cancer progression
Characterization of biventricular alterations in myocardial (reverse) remodelling in aortic banding-induced chronic pressure overload
Montelukast, an available and safe anti-asthmatic drug, prevents maladaptive remodelling and maintains cardiac functionality following myocardial infarction