ANTXR1 deficiency promotes fibroblast senescence: implications for GAPO syndrome as a progeroid disorder

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Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: 1 Online-Ressource.

Language: Englisch

Bibliographic citation: ANTXR1 deficiency promotes fibroblast senescence: implications for GAPO syndrome as a progeroid disorder ; volume:14 ; number:1 ; day:23 ; month:4 ; year:2024 ; pages:1-14 ; date:12.2024
Scientific reports ; 14, Heft 1 (23.4.2024), 1-14, 12.2024

Creator: Przyklenk, Matthias
Karmacharya, Shreya
Bonasera, Debora
Pasanen-Zentz, Arthur-Lauri
Kmoch, Stanislav
Paulsson, Mats
Wagener, Raimund
Liccardi, Gianmaria
Schiavinato, Alvise

Contributor: SpringerLink (Online service)

DOI: 10.1038/s41598-024-59901-y

URN: urn:nbn:de:101:1-2407090910349.507604246734

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:45 AM CEST

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Associated

Przyklenk, Matthias
Karmacharya, Shreya
Bonasera, Debora
Pasanen-Zentz, Arthur-Lauri
Kmoch, Stanislav
Paulsson, Mats
Wagener, Raimund
Liccardi, Gianmaria
Schiavinato, Alvise
SpringerLink (Online service)

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ANTXR1 deficiency promotes fibroblast senescence: implications for GAPO syndrome as a progeroid disorder

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Other Objects (12)

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GATD3A-deficiency-induced mitochondrial dysfunction facilitates senescence of fibroblast-like synoviocytes and osteoarthritis progression

NLRP1 inflammasome promotes senescence and senescence-associated secretory phenotype

p52 signaling promotes cellular senescence

The caspase-activated DNase promotes cellular senescence

Bmi-1 alleviates adventitial fibroblast senescence by eliminating ROS in pulmonary hypertension

The induction of the fibroblast extracellular senescence metabolome is a dynamic process

TET2 deficiency promotes MDS-associated leukemogenesis

Oxidative glucose metabolism promotes senescence in vascular endothelial cells

DNA damage-induced degradation of Sp1 promotes cellular senescence

DNA-PKcs modulates progenitor cell proliferation and fibroblast senescence in idiopathic pulmonary fibrosis

FBXL4 deficiency promotes mitophagy by elevating NIX

Clusterin Deficiency Promotes Cellular Senescence in Human Astrocytes

GATD3A-deficiency-induced mitochondrial dysfunction facilitates senescence of fibroblast-like synoviocytes and osteoarthritis progression

NLRP1 inflammasome promotes senescence and senescence-associated secretory phenotype

p52 signaling promotes cellular senescence

The caspase-activated DNase promotes cellular senescence

Bmi-1 alleviates adventitial fibroblast senescence by eliminating ROS in pulmonary hypertension

The induction of the fibroblast extracellular senescence metabolome is a dynamic process

TET2 deficiency promotes MDS-associated leukemogenesis

Oxidative glucose metabolism promotes senescence in vascular endothelial cells

DNA damage-induced degradation of Sp1 promotes cellular senescence

DNA-PKcs modulates progenitor cell proliferation and fibroblast senescence in idiopathic pulmonary fibrosis

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p52 signaling promotes cellular senescence

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The induction of the fibroblast extracellular senescence metabolome is a dynamic process

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DNA damage-induced degradation of Sp1 promotes cellular senescence

DNA-PKcs modulates progenitor cell proliferation and fibroblast senescence in idiopathic pulmonary fibrosis

FBXL4 deficiency promotes mitophagy by elevating NIX

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