FOXM1 modulates docetaxel resistance in prostate cancer by regulating KIF20A
- Standort
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1475-2867
- Umfang
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Online-Ressource
- Sprache
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Englisch
- Anmerkungen
-
online resource.
- Erschienen in
-
FOXM1 modulates docetaxel resistance in prostate cancer by regulating KIF20A ; volume:20 ; number:1 ; day:10 ; month:11 ; year:2020 ; pages:1-12 ; date:12.2020
Cancer cell international ; 20, Heft 1 (10.11.2020), 1-12, 12.2020
- Urheber
-
Yu, Hongbo
Xu, Zheng
Guo, Maomao
Wang, Weiwan
Zhang, Weican
Liang, Sudong
Xu, Zhibin
Ye, Jun
Zhu, Gangyi
Zhang, Chenyang
Lin, Jianzhong
- Beteiligte Personen und Organisationen
-
SpringerLink (Online service)
- DOI
-
10.1186/s12935-020-01631-y
- URN
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urn:nbn:de:101:1-2020122119452216236639
- Rechteinformation
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
- 15.08.2025, 07:35 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
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Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Yu, Hongbo
- Xu, Zheng
- Guo, Maomao
- Wang, Weiwan
- Zhang, Weican
- Liang, Sudong
- Xu, Zhibin
- Ye, Jun
- Zhu, Gangyi
- Zhang, Chenyang
- Lin, Jianzhong
- SpringerLink (Online service)
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Computer-aided analysis of quercetin mechanism of overcoming docetaxel resistance in docetaxel-resistant prostate cancer
GLIPR1-ΔTM synergizes with docetaxel in cell death and suppresses resistance to docetaxel in prostate cancer cells
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FOXM1 modulates 5-FU resistance in colorectal cancer through regulating TYMS expression
FOXM1 promotes hepatocellular carcinoma progression by regulating KIF4A expression
SR9009 inhibits lethal prostate cancer subtype 1 by regulating the LXRα/FOXM1 pathway independently of REV-ERBs
Role of FOXM1 and AURKB in regulating keratinocyte function in psoriasis
FOXM1 modulates 5-fluorouracil sensitivity in cholangiocarcinoma through thymidylate synthase (TYMS): implications of FOXM1–TYMS axis uncoupling in 5-FU resistance
FOXM1 contributes to taxane resistance by regulating UHRF1-controlled cancer cell stemness
Computer-aided analysis of quercetin mechanism of overcoming docetaxel resistance in docetaxel-resistant prostate cancer
GLIPR1-ΔTM synergizes with docetaxel in cell death and suppresses resistance to docetaxel in prostate cancer cells
Tau oligomers accumulation sensitizes prostate cancer cells to docetaxel treatment
SNHG1 opposes quiescence and promotes docetaxel sensitivity in prostate cancer
Glucocorticoid receptor antagonism reverts docetaxel resistance in human prostate cancer