Progression of albuminuria and podocyte injury in focal segmental glomerulosclerosis inhibited by enhanced glycosphingolipid GM3 via valproic acid
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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1 Online-Ressource.
- Language
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Englisch
- Bibliographic citation
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Progression of albuminuria and podocyte injury in focal segmental glomerulosclerosis inhibited by enhanced glycosphingolipid GM3 via valproic acid ; volume:13 ; number:1 ; day:15 ; month:12 ; year:2023 ; pages:1-10 ; date:12.2023
Scientific reports ; 13, Heft 1 (15.12.2023), 1-10, 12.2023
- Creator
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Kawashima, Nagako
Naito, Shokichi
Nagane, Masaki
Yamashita, Tadashi
Nakayama, Ken-ichi
- Contributor
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SpringerLink (Online service)
- DOI
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10.1038/s41598-023-49684-z
- URN
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urn:nbn:de:101:1-2024030221152355834951
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:45 AM CEST
Data provider
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Kawashima, Nagako
- Naito, Shokichi
- Nagane, Masaki
- Yamashita, Tadashi
- Nakayama, Ken-ichi
- SpringerLink (Online service)
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Therapeutic Potential Targeting Podocyte Mitochondrial Dysfunction in Focal Segmental Glomerulosclerosis
Btg2 Promotes Focal Segmental Glomerulosclerosis via Smad3‐Dependent Podocyte‐Mesenchymal Transition
Lysyl oxidase-like 2 inhibition ameliorates glomerulosclerosis and albuminuria in diabetic nephropathy
Pharmacogenetic podocyte depletion in zebrafish larvae as a model for focal segmental glomerulosclerosis
Acatalasemic mice are mildly susceptible to adriamycin nephropathy and exhibit increased albuminuria and glomerulosclerosis
B7-1 mediates podocyte injury and glomerulosclerosis through communication with Hsp90ab1-LRP5-β-catenin pathway
Assessment of podocyte detachment as a pivotal step in the development of focal segmental glomerulosclerosis
Gold nanoparticles attenuate albuminuria by inhibiting podocyte injury in a rat model of diabetic nephropathy
Nuclear exclusion of YAP exacerbates podocyte apoptosis and disease progression in Adriamycin-induced focal segmental glomerulosclerosis
Angiopoietin-like-3 knockout protects against glomerulosclerosis in murine adriamycin-induced nephropathy by attenuating podocyte loss
Revealing the molecular nuances of TRPC6 in focal segmental glomerulosclerosis in an in vitro human podocyte model
Deletion of podocyte Rho-associated, coiled-coil-containing protein kinase 2 protects mice from focal segmental glomerulosclerosis
Therapeutic Potential Targeting Podocyte Mitochondrial Dysfunction in Focal Segmental Glomerulosclerosis
Btg2 Promotes Focal Segmental Glomerulosclerosis via Smad3‐Dependent Podocyte‐Mesenchymal Transition
Lysyl oxidase-like 2 inhibition ameliorates glomerulosclerosis and albuminuria in diabetic nephropathy
Pharmacogenetic podocyte depletion in zebrafish larvae as a model for focal segmental glomerulosclerosis
Acatalasemic mice are mildly susceptible to adriamycin nephropathy and exhibit increased albuminuria and glomerulosclerosis
B7-1 mediates podocyte injury and glomerulosclerosis through communication with Hsp90ab1-LRP5-β-catenin pathway
Assessment of podocyte detachment as a pivotal step in the development of focal segmental glomerulosclerosis
Gold nanoparticles attenuate albuminuria by inhibiting podocyte injury in a rat model of diabetic nephropathy
Nuclear exclusion of YAP exacerbates podocyte apoptosis and disease progression in Adriamycin-induced focal segmental glomerulosclerosis
Angiopoietin-like-3 knockout protects against glomerulosclerosis in murine adriamycin-induced nephropathy by attenuating podocyte loss
Revealing the molecular nuances of TRPC6 in focal segmental glomerulosclerosis in an in vitro human podocyte model