Supplementation with a selective amino acid formula ameliorates muscular dystrophy in mdx mice
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
2045-2322
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Supplementation with a selective amino acid formula ameliorates muscular dystrophy in mdx mice ; volume:8 ; number:1 ; day:2 ; month:10 ; year:2018 ; pages:1-17 ; date:12.2018
Scientific reports ; 8, Heft 1 (2.10.2018), 1-17, 12.2018
- Creator
-
Banfi, Stefania
- Contributor
-
D’Antona, Giuseppe
Ruocco, Chiara
Meregalli, Mirella
Belicchi, Marzia
Bella, Pamela
Erratico, Silvia
Donato, Elisa
Rossi, Fabio
Bifari, Francesco
Lonati, Caterina
Campaner, Stefano
Nisoli, Enzo
Torrente, Yvan
SpringerLink (Online service)
- DOI
-
10.1038/s41598-018-32613-w
- URN
-
urn:nbn:de:101:1-2018111015341180470618
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:53 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Banfi, Stefania
- D’Antona, Giuseppe
- Ruocco, Chiara
- Meregalli, Mirella
- Belicchi, Marzia
- Bella, Pamela
- Erratico, Silvia
- Donato, Elisa
- Rossi, Fabio
- Bifari, Francesco
- Lonati, Caterina
- Campaner, Stefano
- Nisoli, Enzo
- Torrente, Yvan
- SpringerLink (Online service)
Other Objects (12)
Inactivation of Sirt6 ameliorates muscular dystrophy in mdx mice by releasing suppression of utrophin expression
miR-146a deficiency does not aggravate muscular dystrophy in mdx mice
Moderate exercise improves function and increases adiponectin in the mdx mouse model of muscular dystrophy
Proteomic profiling of liver tissue from the mdx-4cv mouse model of Duchenne muscular dystrophy
Biomarkers for Duchenne muscular dystrophy progression: impact of age in the mdx tongue spared muscle
Blockade of IGF2R improves muscle regeneration and ameliorates Duchenne muscular dystrophy
Inactivation of the protein deacetylase SIRT6 ameliorates muscular dystrophy in mdx mice by upregulation of utrophin and normalization of asymmetric muscle stem cell division
Pannexin 1 dysregulation in Duchenne muscular dystrophy and its exacerbation of dystrophic features in mdx mice
Increased plasma lipid levels exacerbate muscle pathology in the mdx mouse model of Duchenne muscular dystrophy
Myelination is delayed during postnatal brain development in the mdx mouse model of Duchenne muscular dystrophy
Muscular dystrophy in the mdx mouse is a severe myopathy compounded by hypotrophy, hypertrophy and hyperplasia
Altered social behavior and ultrasonic communication in the dystrophin-deficient mdx mouse model of Duchenne muscular dystrophy
Inactivation of Sirt6 ameliorates muscular dystrophy in mdx mice by releasing suppression of utrophin expression
miR-146a deficiency does not aggravate muscular dystrophy in mdx mice
Moderate exercise improves function and increases adiponectin in the mdx mouse model of muscular dystrophy
Proteomic profiling of liver tissue from the mdx-4cv mouse model of Duchenne muscular dystrophy
Biomarkers for Duchenne muscular dystrophy progression: impact of age in the mdx tongue spared muscle
Blockade of IGF2R improves muscle regeneration and ameliorates Duchenne muscular dystrophy
Inactivation of the protein deacetylase SIRT6 ameliorates muscular dystrophy in mdx mice by upregulation of utrophin and normalization of asymmetric muscle stem cell division
Pannexin 1 dysregulation in Duchenne muscular dystrophy and its exacerbation of dystrophic features in mdx mice
Increased plasma lipid levels exacerbate muscle pathology in the mdx mouse model of Duchenne muscular dystrophy
Myelination is delayed during postnatal brain development in the mdx mouse model of Duchenne muscular dystrophy
Muscular dystrophy in the mdx mouse is a severe myopathy compounded by hypotrophy, hypertrophy and hyperplasia
Altered social behavior and ultrasonic communication in the dystrophin-deficient mdx mouse model of Duchenne muscular dystrophy
Inactivation of Sirt6 ameliorates muscular dystrophy in mdx mice by releasing suppression of utrophin expression
miR-146a deficiency does not aggravate muscular dystrophy in mdx mice
Moderate exercise improves function and increases adiponectin in the mdx mouse model of muscular dystrophy
Proteomic profiling of liver tissue from the mdx-4cv mouse model of Duchenne muscular dystrophy
Biomarkers for Duchenne muscular dystrophy progression: impact of age in the mdx tongue spared muscle
Blockade of IGF2R improves muscle regeneration and ameliorates Duchenne muscular dystrophy
Inactivation of the protein deacetylase SIRT6 ameliorates muscular dystrophy in mdx mice by upregulation of utrophin and normalization of asymmetric muscle stem cell division
Pannexin 1 dysregulation in Duchenne muscular dystrophy and its exacerbation of dystrophic features in mdx mice
Increased plasma lipid levels exacerbate muscle pathology in the mdx mouse model of Duchenne muscular dystrophy
Myelination is delayed during postnatal brain development in the mdx mouse model of Duchenne muscular dystrophy
Muscular dystrophy in the mdx mouse is a severe myopathy compounded by hypotrophy, hypertrophy and hyperplasia