Transposable elements activation triggers necroptosis in mouse embryonic stem cells
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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1 Online-Ressource.
- Language
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Englisch
- Bibliographic citation
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Transposable elements activation triggers necroptosis in mouse embryonic stem cells ; volume:14 ; number:3 ; day:7 ; month:3 ; year:2023 ; pages:1-10 ; date:3.2023
Cell death & disease ; 14, Heft 3 (7.3.2023), 1-10, 3.2023
- Creator
-
Jin, Lingmei
He, Jiangping
Feng, Huijian
Li, Sa
Liu, He
Dong, Hongzhi
Hu, MingLi
Huang, Junju
Wu, Haoyu
Chen, Jiekai
Qi, Ling
Wu, Kaixin
- Contributor
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SpringerLink (Online service)
- DOI
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10.1038/s41419-023-05705-3
- URN
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urn:nbn:de:101:1-2410170901244.532316518159
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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15.08.2025, 7:39 AM CEST
Data provider
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Jin, Lingmei
- He, Jiangping
- Feng, Huijian
- Li, Sa
- Liu, He
- Dong, Hongzhi
- Hu, MingLi
- Huang, Junju
- Wu, Haoyu
- Chen, Jiekai
- Qi, Ling
- Wu, Kaixin
- SpringerLink (Online service)
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Tissue-specific usage of transposable element-derived promoters in mouse development
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Differentiation roadmap of embryonic Sertoli cells derived from mouse embryonic stem cells
The role of MORC3 in silencing transposable elements in mouse embryonic stem cells
Transposable elements are regulated by context-specific patterns of chromatin marks in mouse embryonic stem cells
Evolution of mouse circadian enhancers from transposable elements
RIPK1 can mediate apoptosis in addition to necroptosis during embryonic development
Cdk8 and Hira mutations trigger X chromosome elimination in naive female hybrid mouse embryonic stem cells
ZBP1 and TRIF trigger lethal necroptosis in mice lacking caspase-8 and TNFR1
Osteocalcin triggers Fas/FasL-mediated necroptosis in adipocytes via activation of p300
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Aβ oligomers trigger necroptosis-mediated neurodegeneration via microglia activation in Alzheimer’s disease
Tissue-specific usage of transposable element-derived promoters in mouse development
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