- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
2397-768X
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Co-targeting KRAS G12C and EGFR reduces both mutant and wild-type RAS-GTP ; volume:6 ; number:1 ; day:23 ; month:11 ; year:2022 ; pages:1-7 ; date:12.2022
npj precision oncology ; 6, Heft 1 (23.11.2022), 1-7, 12.2022
- Creator
-
McFall, Thomas
Trogdon, Michael
Guizar, Anita C.
Langenheim, John F.
Sisk-Hackworth, Laura
Stites, Edward C.
- Contributor
-
SpringerLink (Online service)
- DOI
-
10.1038/s41698-022-00329-w
- URN
-
urn:nbn:de:101:1-2023020508591484021493
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:56 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- McFall, Thomas
- Trogdon, Michael
- Guizar, Anita C.
- Langenheim, John F.
- Sisk-Hackworth, Laura
- Stites, Edward C.
- SpringerLink (Online service)
Other Objects (12)
Concurrent inhibition of oncogenic and wild-type RAS-GTP for cancer therapy
The relationship between oncogenic KRas and wild-type Ras activity and signaling
Biweekly cetuximab and first-line chemotherapy in chinese patients with k-ras wild-type colorectal cancers
K-Ras protein as a drug target
Unraveling (K)RAS in pancreatic ductal adenocarcinoma
Untersuchung der GAP-katalysierten GTP-Hydrolysereaktion von Ras und Rap
Tumour-selective activity of RAS-GTP inhibition in pancreatic cancer
Untersuchung der GAP-katalysierten GTP-Hydrolysereaktion von Ras und Rap
Excited-state observation of active K-Ras reveals differential structural dynamics of wild-type versus oncogenic G12D and G12C mutants
Restoration of mutant K-Ras repressed miR-199b inhibits K-Ras mutant non-small cell lung cancer progression
K-Ras prenylation as a potential anticancer target
MEK inhibition suppresses K-Ras wild-type cholangiocarcinoma in vitro and in vivo via inhibiting cell proliferation and modulating tumor microenvironment
Concurrent inhibition of oncogenic and wild-type RAS-GTP for cancer therapy
The relationship between oncogenic KRas and wild-type Ras activity and signaling
Biweekly cetuximab and first-line chemotherapy in chinese patients with k-ras wild-type colorectal cancers
K-Ras protein as a drug target
Unraveling (K)RAS in pancreatic ductal adenocarcinoma
Untersuchung der GAP-katalysierten GTP-Hydrolysereaktion von Ras und Rap
Tumour-selective activity of RAS-GTP inhibition in pancreatic cancer
Untersuchung der GAP-katalysierten GTP-Hydrolysereaktion von Ras und Rap
Excited-state observation of active K-Ras reveals differential structural dynamics of wild-type versus oncogenic G12D and G12C mutants
Restoration of mutant K-Ras repressed miR-199b inhibits K-Ras mutant non-small cell lung cancer progression
K-Ras prenylation as a potential anticancer target
MEK inhibition suppresses K-Ras wild-type cholangiocarcinoma in vitro and in vivo via inhibiting cell proliferation and modulating tumor microenvironment
Concurrent inhibition of oncogenic and wild-type RAS-GTP for cancer therapy
The relationship between oncogenic KRas and wild-type Ras activity and signaling
Biweekly cetuximab and first-line chemotherapy in chinese patients with k-ras wild-type colorectal cancers
K-Ras protein as a drug target
Unraveling (K)RAS in pancreatic ductal adenocarcinoma
Untersuchung der GAP-katalysierten GTP-Hydrolysereaktion von Ras und Rap
Tumour-selective activity of RAS-GTP inhibition in pancreatic cancer
Untersuchung der GAP-katalysierten GTP-Hydrolysereaktion von Ras und Rap
Excited-state observation of active K-Ras reveals differential structural dynamics of wild-type versus oncogenic G12D and G12C mutants
Restoration of mutant K-Ras repressed miR-199b inhibits K-Ras mutant non-small cell lung cancer progression
K-Ras prenylation as a potential anticancer target