CD36 aggravates podocyte injury by activating NLRP3 inflammasome and inhibiting autophagy in lupus nephritis
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2041-4889
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
-
online resource.
- Bibliographic citation
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CD36 aggravates podocyte injury by activating NLRP3 inflammasome and inhibiting autophagy in lupus nephritis ; volume:13 ; number:8 ; day:23 ; month:8 ; year:2022 ; pages:1-9 ; date:8.2022
Cell death & disease ; 13, Heft 8 (23.8.2022), 1-9, 8.2022
- Creator
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Lv, Fu
He, Yingxin
Xu, Hongde
Li, Yongchun
Han, Lipei
Yan, Lijie
Lang, Hui
Zhao, Yafei
Zhao, Zhanzheng
Qi, Yuanyuan
- Contributor
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SpringerLink (Online service)
- DOI
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10.1038/s41419-022-05179-9
- URN
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urn:nbn:de:101:1-2022111121031950994728
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:29 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Lv, Fu
- He, Yingxin
- Xu, Hongde
- Li, Yongchun
- Han, Lipei
- Yan, Lijie
- Lang, Hui
- Zhao, Yafei
- Zhao, Zhanzheng
- Qi, Yuanyuan
- SpringerLink (Online service)
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CD36 deficiency affects depressive-like behaviors possibly by modifying gut microbiota and the inflammasome pathway in mice
HOXA11-OS participates in lupus nephritis by targeting miR-124-3p mediating Cyr61 to regulate podocyte autophagy
Activation of Cyclooxygenase-2 by ATF4 During Endoplasmic Reticulum Stress Regulates Kidney Podocyte Autophagy Induced by Lupus Nephritis
Roryt+ ILCs promote crystal-induced nephritis and fibrosis in response to NLRP3-dependet inflammasome activation
CD36-mediated podocyte lipotoxicity promotes foot process effacement
Inducible ATF3–NFAT axis aggravates podocyte injury
Inflammasome activation aggravates choroidal neovascularization
A KDM6A–KLF10 reinforcing feedback mechanism aggravates diabetic podocyte dysfunction
Indoxyl Sulfate Aggravates Podocyte Damage through the TGF-β1/Smad/ROS Signalling Pathway
Acute interstitial nephritis with podocyte foot-process effacement complicating Plasmodium falciparum infection
Deficiency of Nuclear Receptor Coactivator 3 Aggravates Diabetic Kidney Disease by Impairing Podocyte Autophagy
SIRT1 Alleviates Aldosterone-Induced Podocyte Injury by Suppressing Mitochondrial Dysfunction and NLRP3 Inflammasome Activation
CD36 deficiency affects depressive-like behaviors possibly by modifying gut microbiota and the inflammasome pathway in mice
HOXA11-OS participates in lupus nephritis by targeting miR-124-3p mediating Cyr61 to regulate podocyte autophagy
Activation of Cyclooxygenase-2 by ATF4 During Endoplasmic Reticulum Stress Regulates Kidney Podocyte Autophagy Induced by Lupus Nephritis
Roryt+ ILCs promote crystal-induced nephritis and fibrosis in response to NLRP3-dependet inflammasome activation
CD36-mediated podocyte lipotoxicity promotes foot process effacement
Inducible ATF3–NFAT axis aggravates podocyte injury
Inflammasome activation aggravates choroidal neovascularization
A KDM6A–KLF10 reinforcing feedback mechanism aggravates diabetic podocyte dysfunction
Indoxyl Sulfate Aggravates Podocyte Damage through the TGF-β1/Smad/ROS Signalling Pathway
Acute interstitial nephritis with podocyte foot-process effacement complicating Plasmodium falciparum infection
Deficiency of Nuclear Receptor Coactivator 3 Aggravates Diabetic Kidney Disease by Impairing Podocyte Autophagy
SIRT1 Alleviates Aldosterone-Induced Podocyte Injury by Suppressing Mitochondrial Dysfunction and NLRP3 Inflammasome Activation
CD36 deficiency affects depressive-like behaviors possibly by modifying gut microbiota and the inflammasome pathway in mice
HOXA11-OS participates in lupus nephritis by targeting miR-124-3p mediating Cyr61 to regulate podocyte autophagy
Activation of Cyclooxygenase-2 by ATF4 During Endoplasmic Reticulum Stress Regulates Kidney Podocyte Autophagy Induced by Lupus Nephritis