PHF6 recruits BPTF to promote HIF-dependent pathway and progression in YAP-high breast cancer
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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PHF6 recruits BPTF to promote HIF-dependent pathway and progression in YAP-high breast cancer ; volume:21 ; number:1 ; day:26 ; month:3 ; year:2023 ; pages:1-18 ; date:12.2023
Journal of translational medicine ; 21, Heft 1 (26.3.2023), 1-18, 12.2023
- Creator
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Gao, Sheng
Zhang, Wensheng
Ma, Jingjing
Ni, Xiaojian
- Contributor
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SpringerLink (Online service)
- DOI
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10.1186/s12967-023-04031-8
- URN
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urn:nbn:de:101:1-2023081310462484545432
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:55 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Gao, Sheng
- Zhang, Wensheng
- Ma, Jingjing
- Ni, Xiaojian
- SpringerLink (Online service)
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Correction to: PRMT1-mediated H4R3me2a recruits SMARCA4 to promote colorectal cancer progression by enhancing EGFR signaling
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The hypoxia conditioned mesenchymal stem cells promote hepatocellular carcinoma progression through YAP mediated lipogenesis reprogramming
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Cancer cell adaptation to hypoxia involves a HIF‐GPRC5A‐YAP axis
YAP-independent mechanotransduction drives breast cancer progression
HIF factors cooperate with PML ‐ RAR α to promote acute promyelocytic leukemia progression and relapse
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PRMT1-mediated H4R3me2a recruits SMARCA4 to promote colorectal cancer progression by enhancing EGFR signaling
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Yes-associated protein (YAP) binds to HIF-1α and sustains HIF-1α protein stability to promote hepatocellular carcinoma cell glycolysis under hypoxic stress
RUNX2 recruits the NuRD(MTA1)/CRL4B complex to promote breast cancer progression and bone metastasis
Correction to: PRMT1-mediated H4R3me2a recruits SMARCA4 to promote colorectal cancer progression by enhancing EGFR signaling
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The hypoxia conditioned mesenchymal stem cells promote hepatocellular carcinoma progression through YAP mediated lipogenesis reprogramming
Author Correction: Dysfunctional adipocytes promote tumor progression through YAP/TAZ-dependent cancer-associated adipocyte transformation
Cancer cell adaptation to hypoxia involves a HIF‐GPRC5A‐YAP axis
YAP-independent mechanotransduction drives breast cancer progression