Defining the relative and combined contribution of CTCF and CTCFL to genomic regulation
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1474-760X
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Defining the relative and combined contribution of CTCF and CTCFL to genomic regulation ; volume:21 ; number:1 ; day:11 ; month:5 ; year:2020 ; pages:1-34 ; date:12.2020
Genome biology ; 21, Heft 1 (11.5.2020), 1-34, 12.2020
- Creator
-
Nishana, Mayilaadumveettil
Ha, Caryn
Rodriguez-Hernaez, Javier
Ranjbaran, Ali
Chio, Erica
Nora, Elphege P.
Badri, Sana B.
Kloetgen, Andreas
Bruneau, Benoit G.
Tsirigos, Aristotelis
Skok, Jane A.
- Contributor
-
SpringerLink (Online service)
- DOI
-
10.1186/s13059-020-02024-0
- URN
-
urn:nbn:de:101:1-2020080320371421021593
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:51 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Nishana, Mayilaadumveettil
- Ha, Caryn
- Rodriguez-Hernaez, Javier
- Ranjbaran, Ali
- Chio, Erica
- Nora, Elphege P.
- Badri, Sana B.
- Kloetgen, Andreas
- Bruneau, Benoit G.
- Tsirigos, Aristotelis
- Skok, Jane A.
- SpringerLink (Online service)
Other Objects (12)
Author Correction: Defining the relative and combined contribution of CTCF and CTCFL to genomic regulation
Predicting CTCF-mediated chromatin interactions by integrating genomic and epigenomic features
CTCF binding site classes exhibit distinct evolutionary, genomic, epigenomic and transcriptomic features
CTCF and cellular heterogeneity
Defining the genomic signature of the parous breast
Genomic imbalances defining novel intellectual disability associated loci
Genomic features defining exonic variants that modulate splicing
Funktionen des multivalenten Transkriptionsfaktors CTCF
Genomic retargeting of p53 and CTCF is associated with transcriptional changes during oncogenic HRas-induced transformation
An alternative CTCF isoform antagonizes canonical CTCF occupancy and changes chromatin architecture to promote apoptosis
Identifying cancer drivers at CTCF insulators
CGGBP1 regulates CTCF occupancy at repeats
Author Correction: Defining the relative and combined contribution of CTCF and CTCFL to genomic regulation
Predicting CTCF-mediated chromatin interactions by integrating genomic and epigenomic features
CTCF binding site classes exhibit distinct evolutionary, genomic, epigenomic and transcriptomic features
CTCF and cellular heterogeneity
Defining the genomic signature of the parous breast
Genomic imbalances defining novel intellectual disability associated loci
Genomic features defining exonic variants that modulate splicing
Funktionen des multivalenten Transkriptionsfaktors CTCF
Genomic retargeting of p53 and CTCF is associated with transcriptional changes during oncogenic HRas-induced transformation
An alternative CTCF isoform antagonizes canonical CTCF occupancy and changes chromatin architecture to promote apoptosis
Identifying cancer drivers at CTCF insulators
CGGBP1 regulates CTCF occupancy at repeats
Author Correction: Defining the relative and combined contribution of CTCF and CTCFL to genomic regulation
Predicting CTCF-mediated chromatin interactions by integrating genomic and epigenomic features
CTCF binding site classes exhibit distinct evolutionary, genomic, epigenomic and transcriptomic features
CTCF and cellular heterogeneity
Defining the genomic signature of the parous breast
Genomic imbalances defining novel intellectual disability associated loci
Genomic features defining exonic variants that modulate splicing
Funktionen des multivalenten Transkriptionsfaktors CTCF
Genomic retargeting of p53 and CTCF is associated with transcriptional changes during oncogenic HRas-induced transformation
An alternative CTCF isoform antagonizes canonical CTCF occupancy and changes chromatin architecture to promote apoptosis
Identifying cancer drivers at CTCF insulators