Author Correction: ASS1 metabolically contributes to the nuclear and cytosolic p53-mediated DNA damage response
- Standort
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Deutsche Nationalbibliothek Frankfurt am Main
- Umfang
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1 Online-Ressource.
- Sprache
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Englisch
- Erschienen in
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Author Correction: ASS1 metabolically contributes to the nuclear and cytosolic p53-mediated DNA damage response ; volume:6 ; number:7 ; day:4 ; month:7 ; year:2024 ; pages:1417 ; date:7.2024
Nature metabolism ; 6, Heft 7 (4.7.2024), 1417, 7.2024
- Urheber
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Lim, Lisha Qiu Jin
Adler, Lital
Hajaj, Emma
Soria, Leandro R.
Perry, Rotem Ben-Tov
Darzi, Naama
Brody, Ruchama
Furth, Noa
Lichtenstein, Michal
Bab-Dinitz, Elizabeta
Porat, Ziv
Melman, Tevie
Brandis, Alexander
Malitsky, Sergey
Itkin, Maxim
Aylon, Yael
Ben-Dor, Shifra
Orr, Irit
Pri-Or, Amir
Seger, Rony
Shaul, Yoav
Ruppin, Eytan
Oren, Moshe
Perez, Minervo
Meier, Jordan
Brunetti-Pierri, Nicola
Shema, Efrat
Ulitsky, Igor
Erez, Ayelet
- Beteiligte Personen und Organisationen
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SpringerLink (Online service)
- DOI
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10.1038/s42255-024-01090-z
- URN
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urn:nbn:de:101:1-2410100733100.702175516424
- Rechteinformation
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
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15.08.2025, 07:20 MESZ
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Beteiligte
- Lim, Lisha Qiu Jin
- Adler, Lital
- Hajaj, Emma
- Soria, Leandro R.
- Perry, Rotem Ben-Tov
- Darzi, Naama
- Brody, Ruchama
- Furth, Noa
- Lichtenstein, Michal
- Bab-Dinitz, Elizabeta
- Porat, Ziv
- Melman, Tevie
- Brandis, Alexander
- Malitsky, Sergey
- Itkin, Maxim
- Aylon, Yael
- Ben-Dor, Shifra
- Orr, Irit
- Pri-Or, Amir
- Seger, Rony
- Shaul, Yoav
- Ruppin, Eytan
- Oren, Moshe
- Perez, Minervo
- Meier, Jordan
- Brunetti-Pierri, Nicola
- Shema, Efrat
- Ulitsky, Igor
- Erez, Ayelet
- SpringerLink (Online service)
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ASS1 metabolically contributes to the nuclear and cytosolic p53-mediated DNA damage response
PML contributes to p53-independent p21 up-regulation in gamma-irradiation induced DNA damage responses
p53 Family Proteins : Damage limitation
ASL expression in ALDH1A1+ neurons in the substantia nigra metabolically contributes to neurodegenerative phenotype
Cytosolic phospholipase A2 contributes to innate immune defense against Candida albicans lung infection
Mutant p53 gains oncogenic functions through a chromosomal instability-induced cytosolic DNA response
Cytosolic galectin-7 impairs p53 functions and induces chemoresistance in breast cancer cells
Metabolically induced neuronal differentiation
Constant rate of p53 tetramerization in response to DNA damage controls the p53 response
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BMAL1/p53 mediating bronchial epithelial cell autophagy contributes to PM2.5-aggravated asthma
ASS1 metabolically contributes to the nuclear and cytosolic p53-mediated DNA damage response
PML contributes to p53-independent p21 up-regulation in gamma-irradiation induced DNA damage responses
p53 Family Proteins : Damage limitation
ASL expression in ALDH1A1+ neurons in the substantia nigra metabolically contributes to neurodegenerative phenotype
Cytosolic phospholipase A2 contributes to innate immune defense against Candida albicans lung infection
Mutant p53 gains oncogenic functions through a chromosomal instability-induced cytosolic DNA response
Cytosolic galectin-7 impairs p53 functions and induces chemoresistance in breast cancer cells
Metabolically induced neuronal differentiation
Constant rate of p53 tetramerization in response to DNA damage controls the p53 response
Alleviation of ultraviolet-C-induced oxidative damage through overexpression of cytosolic ascorbate peroxidase
CCL3 contributes to secondary damage after spinal cord injury