Development of DuoMYC: a synthetic cell penetrant miniprotein that efficiently inhibits the oncogenic transcription factor MYC
- Standort
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Deutsche Nationalbibliothek Frankfurt am Main
- Umfang
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Online-Ressource
- Sprache
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Englisch
- Erschienen in
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Development of DuoMYC: a synthetic cell penetrant miniprotein that efficiently inhibits the oncogenic transcription factor MYC ; day:06 ; month:11 ; year:2024 ; extent:9
Angewandte Chemie ; (06.11.2024) (gesamt 9)
- Urheber
-
Ellenbroek, Brecht D.
Kahler, Jan Pascal
Arella, Damiano
Lin, Cherina
Jespers, Willem
Züger, Eliane Ann‐Katrin
Drukker, Micha
Pomplun, Sebastian J.
- DOI
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10.1002/ange.202416082
- URN
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urn:nbn:de:101:1-2411061417443.667196730109
- Rechteinformation
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
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15.08.2025, 07:24 MESZ
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Beteiligte
- Ellenbroek, Brecht D.
- Kahler, Jan Pascal
- Arella, Damiano
- Lin, Cherina
- Jespers, Willem
- Züger, Eliane Ann‐Katrin
- Drukker, Micha
- Pomplun, Sebastian J.
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A selective high affinity MYC-binding compound inhibits MYC:MAX interaction and MYC-dependent tumor cell proliferation
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Cordycepin inhibits colon cancer proliferation by suppressing MYC expression
Synthetic lethalities to target a MYC-driven PDAC subtype
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Vitamin B5 supports MYC oncogenic metabolism and tumor progression in breast cancer
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Charakterisierung der Myc-Funktion durch Miz-1 interaktionsdefiziente Myc-Mutanten
Development of DuoMYC: a synthetic cell penetrant miniprotein that efficiently inhibits the oncogenic transcription factor MYC
A selective high affinity MYC-binding compound inhibits MYC:MAX interaction and MYC-dependent tumor cell proliferation
MYC
MYC
MYC
Cordycepin inhibits colon cancer proliferation by suppressing MYC expression
Synthetic lethalities to target a MYC-driven PDAC subtype
THaNX MYC!
MYC RöK
Vitamin B5 supports MYC oncogenic metabolism and tumor progression in breast cancer
Fungal metabolite FR901228 inhibits c-Myc and Fas ligand expression