Influences of amyloid-β and tau on white matter neurite alterations in dementia with Lewy bodies

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Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: 1 Online-Ressource.

Language: Englisch

Bibliographic citation: Influences of amyloid-β and tau on white matter neurite alterations in dementia with Lewy bodies ; volume:10 ; number:1 ; day:3 ; month:4 ; year:2024 ; pages:1-13 ; date:12.2024
npj Parkinson's Disease ; 10, Heft 1 (3.4.2024), 1-13, 12.2024

Creator: Mak, Elijah
Reid, Robert I.
Przybelski, Scott A.
Lesnick, Timothy G.
Schwarz, Christopher G.
Senjem, Matthew L.
Raghavan, Sheelakumari
Vemuri, Prashanthi
Jack, Clifford R.
Min, Hoon Ki
Jain, Manoj K.
Miyagawa, Toji
Forsberg, Leah K.
Fields, Julie A.
Savica, Rodolfo
Graff-Radford, Jonathan
Jones, David T.
Botha, Hugo
Louis, Erik K. St
Knopman, David S.
Ramanan, Vijay K.
Dickson, Dennis W.
Graff-Radford, Neill R.
Ferman, Tanis J.
Petersen, Ronald C.
Lowe, Val J.
Boeve, Bradley F.
O’Brien, John T.
Kantarci, Kejal

Contributor: SpringerLink (Online service)

DOI: 10.1038/s41531-024-00684-4

URN: urn:nbn:de:101:1-2406190939224.351580597500

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 11:02 AM CEST

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Associated

Mak, Elijah
Reid, Robert I.
Przybelski, Scott A.
Lesnick, Timothy G.
Schwarz, Christopher G.
Senjem, Matthew L.
Raghavan, Sheelakumari
Vemuri, Prashanthi
Jack, Clifford R.
Min, Hoon Ki
Jain, Manoj K.
Miyagawa, Toji
Forsberg, Leah K.
Fields, Julie A.
Savica, Rodolfo
Graff-Radford, Jonathan
Jones, David T.
Botha, Hugo
Louis, Erik K. St
Knopman, David S.
Ramanan, Vijay K.
Dickson, Dennis W.
Graff-Radford, Neill R.
Ferman, Tanis J.
Petersen, Ronald C.
Lowe, Val J.
Boeve, Bradley F.
O’Brien, John T.
Kantarci, Kejal
SpringerLink (Online service)

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Influences of amyloid-β and tau on white matter neurite alterations in dementia with Lewy bodies

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Other Objects (12)

The neuropathological diagnosis of Alzheimer’s disease

Cellular and pathological heterogeneity of primary tauopathies

Origins of amyloid-β

Ablation of specific long PDE4D isoforms increases neurite elongation and conveys protection against amyloid-β pathology

Neuropathology and molecular diagnosis of Synucleinopathies

Amyloid-β peptides in cerebrospinal fluid of patients with dementia with Lewy bodies

Selective amyloid-β lowering agents

Development of conformation specific Amyloid-β [Amyloid-beta] antibodies

Proaggregant nuclear factor(s) trigger rapid formation of α-synuclein aggregates in apoptotic neurons

TDP-43 Pathology in Alzheimer’s Disease

C-terminal and full length TDP-43 specie differ according to FTLD-TDP lesion type but not genetic mutation

Evidence of amyloid-β cerebral amyloid angiopathy transmission through neurosurgery

The neuropathological diagnosis of Alzheimer’s disease

Cellular and pathological heterogeneity of primary tauopathies

Origins of amyloid-β

Ablation of specific long PDE4D isoforms increases neurite elongation and conveys protection against amyloid-β pathology

Neuropathology and molecular diagnosis of Synucleinopathies

Amyloid-β peptides in cerebrospinal fluid of patients with dementia with Lewy bodies

Selective amyloid-β lowering agents

Development of conformation specific Amyloid-β [Amyloid-beta] antibodies

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C-terminal and full length TDP-43 specie differ according to FTLD-TDP lesion type but not genetic mutation

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Selective amyloid-β lowering agents

Development of conformation specific Amyloid-β [Amyloid-beta] antibodies

Proaggregant nuclear factor(s) trigger rapid formation of α-synuclein aggregates in apoptotic neurons

TDP-43 Pathology in Alzheimer’s Disease

C-terminal and full length TDP-43 specie differ according to FTLD-TDP lesion type but not genetic mutation

Evidence of amyloid-β cerebral amyloid angiopathy transmission through neurosurgery

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