Cannabidiol ameliorates mitochondrial disease via PPARγ activation in preclinical models
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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1 Online-Ressource.
- Language
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Englisch
- Bibliographic citation
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Cannabidiol ameliorates mitochondrial disease via PPARγ activation in preclinical models ; volume:15 ; number:1 ; day:4 ; month:9 ; year:2024 ; pages:1-21 ; date:12.2024
Nature Communications ; 15, Heft 1 (4.9.2024), 1-21, 12.2024
- Classification
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Medizin, Gesundheit
- Creator
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Puighermanal, Emma
Luna-Sánchez, Marta
Gella, Alejandro
van der Walt, Gunter
Urpi, Andrea
Royo, María
Tena-Morraja, Paula
Appiah, Isabella
de Donato, Maria Helena
Menardy, Fabien
Bianchi, Patrizia
Esteve-Codina, Anna
Rodríguez-Pascau, Laura
Vergara, Cristina
Gómez-Pallarès, Mercè
Marsicano, Giovanni
Bellocchio, Luigi
Martinell, Marc
Sanz, Elisenda
Jurado, Sandra
Soriano, Francesc Xavier
Pizcueta, Pilar
Quintana, Albert
- Contributor
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SpringerLink (Online service)
- DOI
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10.1038/s41467-024-51884-8
- URN
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urn:nbn:de:101:1-2411261257336.741597318121
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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15.08.2025, 7:34 AM CEST
Data provider
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Puighermanal, Emma
- Luna-Sánchez, Marta
- Gella, Alejandro
- van der Walt, Gunter
- Urpi, Andrea
- Royo, María
- Tena-Morraja, Paula
- Appiah, Isabella
- de Donato, Maria Helena
- Menardy, Fabien
- Bianchi, Patrizia
- Esteve-Codina, Anna
- Rodríguez-Pascau, Laura
- Vergara, Cristina
- Gómez-Pallarès, Mercè
- Marsicano, Giovanni
- Bellocchio, Luigi
- Martinell, Marc
- Sanz, Elisenda
- Jurado, Sandra
- Soriano, Francesc Xavier
- Pizcueta, Pilar
- Quintana, Albert
- SpringerLink (Online service)
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Inhibition of mitochondrial OMA1 ameliorates osteosarcoma tumorigenesis
PPARγ regulates osteoarthritis chondrocytes apoptosis through caspase-3 dependent mitochondrial pathway
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A synthetic PPAR-γ agonist triterpenoid ameliorates experimental fibrosis: PPAR-γ-independent suppression of fibrotic responses
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