Parasite proliferation and host cell phenotype drive Leishmania exit from infected macrophages
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
Mainz, Johannes Gutenberg-Universität Mainz, Dissertation, 2024
- Keyword
-
Proliferation
Immunreaktion
Parasitäre Krankheit
Makrophage
- Event
-
Veröffentlichung
- (where)
-
Mainz
- (who)
-
Universitätsbibliothek der Johannes Gutenberg-Universität Mainz
- (when)
-
2024
- Creator
- DOI
-
10.25358/openscience-10932
- URN
-
urn:nbn:de:hebis:77-openscience-5be5073a-6e1a-40ea-9bae-bd759c4e53967
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:38 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Jaedtka, Moritz
- Universitätsbibliothek der Johannes Gutenberg-Universität Mainz
Time of origin
- 2024
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Moroccan strains of Leishmania major and Leishmania tropica differentially impact on nitric oxide production by macrophages
Design of commercially comparable nanotherapeutic agent against human disease-causing parasite, Leishmania
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Two biochemically distinct lipophosphoglycans from Leishmania braziliensis and Leishmania infantum trigger different innate immune responses in murine macrophages
Transcriptional signatures of BALB/c mouse macrophages housing multiplying Leishmania amazonensis amastigotes
Complement receptor 1 mediated control of Leishmania infection in inflammatory human macrophages
Phagocytes and the Leishmania Parasite: A Marriage of Convenience
Alternative hosts of Leishmania infantum: a neglected parasite in Europe
Leishmania major parasites and their interaction with human macrophages
Leishmania major parasites and their interaction with human macrophages
Genome-wide mapping of 5-hydroxymethyluracil in the eukaryote parasite Leishmania
Moroccan strains of Leishmania major and Leishmania tropica differentially impact on nitric oxide production by macrophages
Design of commercially comparable nanotherapeutic agent against human disease-causing parasite, Leishmania
Suppression of LPS-induced inflammatory responses in macrophages infected with Leishmania
Cathelicidin contributes to the restriction of Leishmania in human host macrophages
Two biochemically distinct lipophosphoglycans from Leishmania braziliensis and Leishmania infantum trigger different innate immune responses in murine macrophages
Transcriptional signatures of BALB/c mouse macrophages housing multiplying Leishmania amazonensis amastigotes