Parasite proliferation and host cell phenotype drive Leishmania exit from infected macrophages
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Anmerkungen
-
Mainz, Johannes Gutenberg-Universität Mainz, Dissertation, 2024
- Schlagwort
-
Proliferation
Immunreaktion
Parasitäre Krankheit
Makrophage
- Ereignis
-
Veröffentlichung
- (wo)
-
Mainz
- (wer)
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Universitätsbibliothek der Johannes Gutenberg-Universität Mainz
- (wann)
-
2024
- Urheber
- DOI
-
10.25358/openscience-10932
- URN
-
urn:nbn:de:hebis:77-openscience-5be5073a-6e1a-40ea-9bae-bd759c4e53967
- Rechteinformation
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
15.08.2025, 07:38 MESZ
Datenpartner
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Beteiligte
- Jaedtka, Moritz
- Universitätsbibliothek der Johannes Gutenberg-Universität Mainz
Entstanden
- 2024
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Moroccan strains of Leishmania major and Leishmania tropica differentially impact on nitric oxide production by macrophages
Design of commercially comparable nanotherapeutic agent against human disease-causing parasite, Leishmania
Suppression of LPS-induced inflammatory responses in macrophages infected with Leishmania
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Two biochemically distinct lipophosphoglycans from Leishmania braziliensis and Leishmania infantum trigger different innate immune responses in murine macrophages
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Phagocytes and the Leishmania Parasite: A Marriage of Convenience
Alternative hosts of Leishmania infantum: a neglected parasite in Europe
Leishmania major parasites and their interaction with human macrophages
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Genome-wide mapping of 5-hydroxymethyluracil in the eukaryote parasite Leishmania
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Design of commercially comparable nanotherapeutic agent against human disease-causing parasite, Leishmania
Suppression of LPS-induced inflammatory responses in macrophages infected with Leishmania
Cathelicidin contributes to the restriction of Leishmania in human host macrophages
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Transcriptional signatures of BALB/c mouse macrophages housing multiplying Leishmania amazonensis amastigotes