HDAC8 suppresses the epithelial phenotype and promotes EMT in chemotherapy-treated basal-like breast cancer
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1868-7083
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
-
HDAC8 suppresses the epithelial phenotype and promotes EMT in chemotherapy-treated basal-like breast cancer ; volume:14 ; number:1 ; day:11 ; month:1 ; year:2022 ; pages:1-16 ; date:12.2022
Clinical epigenetics ; 14, Heft 1 (11.1.2022), 1-16, 12.2022
- Creator
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Pantelaiou-Prokaki, Garyfallia
Mieczkowska, Iga
Schmidt, Geske E.
Fritzsche, Sonja
Prokakis, Evangelos
Gallwas, Julia
Wegwitz, Florian
- Contributor
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SpringerLink (Online service)
- DOI
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10.1186/s13148-022-01228-4
- URN
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urn:nbn:de:101:1-2022042520330993575124
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:32 AM CEST
Data provider
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Associated
- Pantelaiou-Prokaki, Garyfallia
- Mieczkowska, Iga
- Schmidt, Geske E.
- Fritzsche, Sonja
- Prokakis, Evangelos
- Gallwas, Julia
- Wegwitz, Florian
- SpringerLink (Online service)
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Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC
WNT5B governs the phenotype of basal-like breast cancer by activating WNT signaling
Basal-like phenotype is not associated with patient survival in estrogen-receptor-negative breast cancers
Feline mammary basal-like adenocarcinomas: a potential model for human triple-negative breast cancer (TNBC) with basal-like subtype
Expression of thyroid transcription factor-1 is associated with a basal-like phenotype in breast carcinomas
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Targeting BCL-2–expressing basal-like breast cancer with BH3-mimetics
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The CD44+/CD24-phenotype is enriched in basal-like breast tumors
Axon guidance cue SEMA3A promotes the aggressive phenotype of basal-like PDAC
WNT5B governs the phenotype of basal-like breast cancer by activating WNT signaling
Basal-like phenotype is not associated with patient survival in estrogen-receptor-negative breast cancers
Feline mammary basal-like adenocarcinomas: a potential model for human triple-negative breast cancer (TNBC) with basal-like subtype
Expression of thyroid transcription factor-1 is associated with a basal-like phenotype in breast carcinomas
Integrin-mediated tumor-stroma interfacial signaling promotes basal-like and immune exclusion phenotype in pancreatic cancer
Inactivation of LATS1/2 drives luminal-basal plasticity to initiate basal-like mammary carcinomas
Weight loss reduces basal-like breast cancer through kinome reprogramming
Basal-like breast cancer: molecular profiles, clinical features and survival outcomes