Autism-associated mutations interfere with the function of the Shank3 N-terminus in postsynaptic signaling pathways
- Alternative title
-
Autismus-assoziierte Mutationen beeinträchtigen die Funktion des N-Terminus von Shank3 in der postsynaptischen Signaltransduktion
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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Online-Ressource
- Language
-
Englisch
- Notes
-
Hamburg, Universität Hamburg, Dissertation, 2019
- Classification
-
Biowissenschaften, Biologie
- Event
-
Veröffentlichung
- (where)
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Hamburg
- (who)
-
Staats- und Universitätsbibliothek Hamburg
- (when)
-
2019
- Creator
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Hassani Nia, Fatemeh
- Contributor
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Kreienkamp, Hans-Jürgen
- URN
-
urn:nbn:de:gbv:18-97457
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 11:00 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Hassani Nia, Fatemeh
- Kreienkamp, Hans-Jürgen
- Staats- und Universitätsbibliothek Hamburg
Time of origin
- 2019
Other Objects (12)
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Shank3 Deficiency Results in a Reduction in GABAergic Postsynaptic Puncta in the Olfactory Brain Areas
Transcriptional and functional complexity of Shank3 provides a molecular framework to understand the phenotypic heterogeneity of SHANK3 causing autism and Shank3 mutant mice
Autism-associated gene shank3 is necessary for social contagion in zebrafish
The Role of Thioredoxin System in Shank3 Mouse Model of Autism
Association study of SHANK3 gene polymorphisms with autism in Chinese Han population
Disrupted Human–Dog Interbrain Neural Coupling in Autism‐Associated Shank3 Mutant Dogs
Combined expansion and STED microscopy reveals altered fingerprints of postsynaptic nanostructure across brain regions in ASD-related SHANK3-deficiency
Single-molecule fluorescence in-situ hybridization reveals that human SHANK3 mRNA expression varies during development and in autism-associated SHANK3 heterozygosity
Characterization of the structural and functional impact of autism-associated SHANK3 missense mutations
Sex differences in sleep deficits in mice with an autism-linked Shank3 mutation
Targeting of δ-catenin to postsynaptic sites through interaction with the Shank3 N-terminus
The Shank/ProSAP N-Terminal (SPN) Domain of Shank3 Regulates Targeting to Postsynaptic Sites and Postsynaptic Signaling
Shank3 Deficiency Results in a Reduction in GABAergic Postsynaptic Puncta in the Olfactory Brain Areas
Transcriptional and functional complexity of Shank3 provides a molecular framework to understand the phenotypic heterogeneity of SHANK3 causing autism and Shank3 mutant mice
Autism-associated gene shank3 is necessary for social contagion in zebrafish
The Role of Thioredoxin System in Shank3 Mouse Model of Autism
Association study of SHANK3 gene polymorphisms with autism in Chinese Han population
Disrupted Human–Dog Interbrain Neural Coupling in Autism‐Associated Shank3 Mutant Dogs
Combined expansion and STED microscopy reveals altered fingerprints of postsynaptic nanostructure across brain regions in ASD-related SHANK3-deficiency
Single-molecule fluorescence in-situ hybridization reveals that human SHANK3 mRNA expression varies during development and in autism-associated SHANK3 heterozygosity
Characterization of the structural and functional impact of autism-associated SHANK3 missense mutations
Sex differences in sleep deficits in mice with an autism-linked Shank3 mutation
Targeting of δ-catenin to postsynaptic sites through interaction with the Shank3 N-terminus
The Shank/ProSAP N-Terminal (SPN) Domain of Shank3 Regulates Targeting to Postsynaptic Sites and Postsynaptic Signaling
Shank3 Deficiency Results in a Reduction in GABAergic Postsynaptic Puncta in the Olfactory Brain Areas
Transcriptional and functional complexity of Shank3 provides a molecular framework to understand the phenotypic heterogeneity of SHANK3 causing autism and Shank3 mutant mice
Autism-associated gene shank3 is necessary for social contagion in zebrafish
The Role of Thioredoxin System in Shank3 Mouse Model of Autism
Association study of SHANK3 gene polymorphisms with autism in Chinese Han population
Disrupted Human–Dog Interbrain Neural Coupling in Autism‐Associated Shank3 Mutant Dogs
Combined expansion and STED microscopy reveals altered fingerprints of postsynaptic nanostructure across brain regions in ASD-related SHANK3-deficiency
Single-molecule fluorescence in-situ hybridization reveals that human SHANK3 mRNA expression varies during development and in autism-associated SHANK3 heterozygosity
Characterization of the structural and functional impact of autism-associated SHANK3 missense mutations