Gambogic acid triggers vacuolization-associated cell death in cancer cells via disruption of thiol proteostasis

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 2041-4889

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Gambogic acid triggers vacuolization-associated cell death in cancer cells via disruption of thiol proteostasis ; volume:10 ; number:3 ; day:22 ; month:2 ; year:2019 ; pages:1-16 ; date:3.2019
Cell death & disease ; 10, Heft 3 (22.2.2019), 1-16, 3.2019

Creator: Seo, Min Ji
Lee, Dong Min
Kim, In Young
Lee, Dongjoo
Choi, Min-Koo
Lee, Joo-Youn
Park, Seok Soon
Jeong, Seong-Yun
Choi, Eun Kyung
Choi, Kyeong Sook

Contributor: SpringerLink (Online service)

DOI: 10.1038/s41419-019-1360-4

URN: urn:nbn:de:101:1-2019032021401507749302

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 10:57 AM CEST

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Associated

Seo, Min Ji
Lee, Dong Min
Kim, In Young
Lee, Dongjoo
Choi, Min-Koo
Lee, Joo-Youn
Park, Seok Soon
Jeong, Seong-Yun
Choi, Eun Kyung
Choi, Kyeong Sook
SpringerLink (Online service)

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Gambogic acid triggers vacuolization-associated cell death in cancer cells via disruption of thiol proteostasis

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Other Objects (12)

KPNB1 inhibition disrupts proteostasis and triggers unfolded protein response-mediated apoptosis in glioblastoma cells

Ageing-dependent thiol oxidation reveals early oxidation of proteins with core proteostasis functions

Mitochondrial disruption resulting from Cepharanthine-mediated TOM inhibition triggers ferroptosis in colorectal cancer cells

Radical‐Mediated Thiol‐Ene Strategy: Photoactivation of Thiol‐Containing Drugs in Cancer Cells

Radical‐Mediated Thiol‐Ene Strategy: Photoactivation of Thiol‐Containing Drugs in Cancer Cells

Overexpression of SIRT3 disrupts mitochondrial proteostasis and cell cycle progression

Oligonucleotide Phosphorothioates Enter Cells by Thiol‐Mediated Uptake

Oligonucleotide Phosphorothioates Enter Cells by Thiol‐Mediated Uptake

Moderate beta-cell ablation triggers synergic compensatory mechanisms even in the absence of overt metabolic disruption

Incomplete cell disruption of resistant microbes

Bax Inhibitor-1 preserves pancreatic β-cell proteostasis by limiting proinsulin misfolding and programmed cell death

Cell clusters softening triggers collective cell migration in vivo

KPNB1 inhibition disrupts proteostasis and triggers unfolded protein response-mediated apoptosis in glioblastoma cells

Ageing-dependent thiol oxidation reveals early oxidation of proteins with core proteostasis functions

Mitochondrial disruption resulting from Cepharanthine-mediated TOM inhibition triggers ferroptosis in colorectal cancer cells

Radical‐Mediated Thiol‐Ene Strategy: Photoactivation of Thiol‐Containing Drugs in Cancer Cells

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Overexpression of SIRT3 disrupts mitochondrial proteostasis and cell cycle progression

Oligonucleotide Phosphorothioates Enter Cells by Thiol‐Mediated Uptake

Oligonucleotide Phosphorothioates Enter Cells by Thiol‐Mediated Uptake

Moderate beta-cell ablation triggers synergic compensatory mechanisms even in the absence of overt metabolic disruption

Incomplete cell disruption of resistant microbes

Bax Inhibitor-1 preserves pancreatic β-cell proteostasis by limiting proinsulin misfolding and programmed cell death

Cell clusters softening triggers collective cell migration in vivo

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Ageing-dependent thiol oxidation reveals early oxidation of proteins with core proteostasis functions

Mitochondrial disruption resulting from Cepharanthine-mediated TOM inhibition triggers ferroptosis in colorectal cancer cells

Radical‐Mediated Thiol‐Ene Strategy: Photoactivation of Thiol‐Containing Drugs in Cancer Cells

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Overexpression of SIRT3 disrupts mitochondrial proteostasis and cell cycle progression

Oligonucleotide Phosphorothioates Enter Cells by Thiol‐Mediated Uptake

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