Mutations in trpγ, the homologue of TRPC6 autism candidate gene, causes autism-like behavioral deficits in Drosophila

zu Verbundenen Objekten

Standort
Deutsche Nationalbibliothek Frankfurt am Main
ISSN
1476-5578
Umfang
Online-Ressource
Sprache
Englisch
Anmerkungen
online resource.

Erschienen in
Mutations in trpγ, the homologue of TRPC6 autism candidate gene, causes autism-like behavioral deficits in Drosophila ; volume:27 ; number:8 ; day:2 ; month:5 ; year:2022 ; pages:3328-3342 ; date:8.2022
Molecular psychiatry ; 27, Heft 8 (2.5.2022), 3328-3342, 8.2022

Urheber
Palacios-Muñoz, Angelina
Paula Moreira, Danielle de
Silva, Valeria
García, Isaac E.
Aboitiz, Francisco
Zarrei, Mehdi
Campos, Gabriele
Rennie, Olivia
Howe, Jennifer L.
Anagnostou, Evdokia
Ambrozewic, Patricia
Scherer, Stephen W.
Passos-Bueno, Maria Rita
Ewer, John
Beteiligte Personen und Organisationen
SpringerLink (Online service)

DOI
10.1038/s41380-022-01555-1
URN
urn:nbn:de:101:1-2023020921193109659431
Rechteinformation
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
Letzte Aktualisierung
14.08.2025, 11:02 MESZ

Datenpartner

Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Original beim Datenpartner anzeigen

Beteiligte

  • Palacios-Muñoz, Angelina
  • Paula Moreira, Danielle de
  • Silva, Valeria
  • García, Isaac E.
  • Aboitiz, Francisco
  • Zarrei, Mehdi
  • Campos, Gabriele
  • Rennie, Olivia
  • Howe, Jennifer L.
  • Anagnostou, Evdokia
  • Ambrozewic, Patricia
  • Scherer, Stephen W.
  • Passos-Bueno, Maria Rita
  • Ewer, John
  • SpringerLink (Online service)

Ähnliche Objekte (12)

Brain hyperserotonemia causes autism-relevant social deficits in mice

Brain hyperserotonemia causes autism-relevant social deficits in mice

Facial expression recognition as a candidate marker for autism spectrum disorder: how frequent and severe are deficits?

Facial expression recognition as a candidate marker for autism spectrum disorder: how frequent and severe are deficits?

Candidate gene study of HOXB1 in autism spectrum disorder

Candidate gene study of HOXB1 in autism spectrum disorder

Prenatal chlorpyrifos leads to autism-like deficits in C57Bl6/J mice

Prenatal chlorpyrifos leads to autism-like deficits in C57Bl6/J mice

Building a protein interaction (PIN) database for autism candidate genes

Building a protein interaction (PIN) database for autism candidate genes

Building a protein interaction (PIN) database for autism candidate genes

Building a protein interaction (PIN) database for autism candidate genes

Identification of candidate intergenic risk loci in autism spectrum disorder

Identification of candidate intergenic risk loci in autism spectrum disorder

Examination of NRCAM, LRRN3, KIAA0716, and LAMB1as autism candidate genes

Examination of NRCAM, LRRN3, KIAA0716, and LAMB1as autism candidate genes

Molecular autism

Zeitschrift

Molecular autism

TAOK2 rescues autism-linked developmental deficits in a 16p11.2 microdeletion mouse model

TAOK2 rescues autism-linked developmental deficits in a 16p11.2 microdeletion mouse model

Attention Deficits Influence the Development of Motor Abnormalities in High Functioning Autism

Attention Deficits Influence the Development of Motor Abnormalities in High Functioning Autism

Atypical brain asymmetry in autism - a candidate for clinically meaningful stratification

Atypical brain asymmetry in autism - a candidate for clinically meaningful stratification

Verbundene Objekte