- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1471-2377
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Anmerkungen
-
online resource.
- Erschienen in
-
Cell stress molecules in the skeletal muscle of GNE myopathy ; volume:13 ; number:1 ; day:12 ; month:3 ; year:2013 ; pages:1-9 ; date:12.2013
BMC neurology ; 13, Heft 1 (12.3.2013), 1-9, 12.2013
- Urheber
-
Fischer, Charlotte
- Beteiligte Personen und Organisationen
-
Kleinschnitz, Konstanze
Wrede, Arne
Muth, Ingrid
Kruse, Niels
Nishino, Ichizo
Schmidt, Jens
SpringerLink (Online service)
- DOI
-
10.1186/1471-2377-13-24
- URN
-
urn:nbn:de:1111-2016030332482
- Rechteinformation
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
- 15.08.2025, 05:20 UTC
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Fischer, Charlotte
- Kleinschnitz, Konstanze
- Wrede, Arne
- Muth, Ingrid
- Kruse, Niels
- Nishino, Ichizo
- Schmidt, Jens
- SpringerLink (Online service)
Ähnliche Objekte (12)
Tropomyosin 3 (TPM3) function in skeletal muscle and in myopathy
Oxidative stress in skeletal muscle
Age-dependent loss of Crls1 causes myopathy and skeletal muscle regeneration failure
Dilated cardiomyopathy-mediated heart failure induces a unique skeletal muscle myopathy with inflammation
Altered skeletal muscle (mitochondrial) properties in patients with mitochondrial DNA single deletion myopathy
L-tyrosine supplementation does not ameliorate skeletal muscle dysfunction in zebrafish and mouse models of dominant skeletal muscle α-actin nemaline myopathy
Skeletal muscle
Skeletal muscle
Author Correction: Age-dependent loss of Crls1 causes myopathy and skeletal muscle regeneration failure
Skeletal muscle delimited myopathy and verapamil toxicity in SUR2 mutant mouse models of AIMS
Nuclear defects in skeletal muscle from a Dynamin 2-linked centronuclear myopathy mouse model
Deletion of the Ste20-like kinase SLK in skeletal muscle results in a progressive myopathy and muscle weakness
Tropomyosin 3 (TPM3) function in skeletal muscle and in myopathy
Oxidative stress in skeletal muscle
Age-dependent loss of Crls1 causes myopathy and skeletal muscle regeneration failure
Dilated cardiomyopathy-mediated heart failure induces a unique skeletal muscle myopathy with inflammation
Altered skeletal muscle (mitochondrial) properties in patients with mitochondrial DNA single deletion myopathy
L-tyrosine supplementation does not ameliorate skeletal muscle dysfunction in zebrafish and mouse models of dominant skeletal muscle α-actin nemaline myopathy
Skeletal muscle
Skeletal muscle
Author Correction: Age-dependent loss of Crls1 causes myopathy and skeletal muscle regeneration failure
Skeletal muscle delimited myopathy and verapamil toxicity in SUR2 mutant mouse models of AIMS
Nuclear defects in skeletal muscle from a Dynamin 2-linked centronuclear myopathy mouse model
Deletion of the Ste20-like kinase SLK in skeletal muscle results in a progressive myopathy and muscle weakness
Tropomyosin 3 (TPM3) function in skeletal muscle and in myopathy
Oxidative stress in skeletal muscle
Age-dependent loss of Crls1 causes myopathy and skeletal muscle regeneration failure
Dilated cardiomyopathy-mediated heart failure induces a unique skeletal muscle myopathy with inflammation
Altered skeletal muscle (mitochondrial) properties in patients with mitochondrial DNA single deletion myopathy
L-tyrosine supplementation does not ameliorate skeletal muscle dysfunction in zebrafish and mouse models of dominant skeletal muscle α-actin nemaline myopathy
Skeletal muscle
Skeletal muscle
Author Correction: Age-dependent loss of Crls1 causes myopathy and skeletal muscle regeneration failure
Skeletal muscle delimited myopathy and verapamil toxicity in SUR2 mutant mouse models of AIMS
Nuclear defects in skeletal muscle from a Dynamin 2-linked centronuclear myopathy mouse model