Prostacyclin reverses platelet stress fibre formation causing platelet aggregate instability
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
2045-2322
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Anmerkungen
-
online resource.
- Erschienen in
-
Prostacyclin reverses platelet stress fibre formation causing platelet aggregate instability ; volume:7 ; number:1 ; day:17 ; month:7 ; year:2017 ; pages:1-11 ; date:12.2017
Scientific reports ; 7, Heft 1 (17.7.2017), 1-11, 12.2017
- Klassifikation
-
Biowissenschaften, Biologie
- Urheber
-
Yusuf, M. Z.
Raslan, Z.
Atkinson, L.
Aburima, A.
Thomas, S. G.
Naseem, K. M.
Calaminus, S. D. J.
- Beteiligte Personen und Organisationen
-
SpringerLink (Online service)
- DOI
-
10.1038/s41598-017-05817-9
- URN
-
urn:nbn:de:101:1-2019061419263755066437
- Rechteinformation
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
14.08.2025, 10:54 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Yusuf, M. Z.
- Raslan, Z.
- Atkinson, L.
- Aburima, A.
- Thomas, S. G.
- Naseem, K. M.
- Calaminus, S. D. J.
- SpringerLink (Online service)
Ähnliche Objekte (12)
What about Platelet Function in Platelet Concentrates?
Platelet immunology
Platelet Glycocalicin
Arachidonic acid metabolite 19(S)-HETE induces vasorelaxation and platelet inhibition by activating prostacyclin (IP) receptor
Platelet Membrane Receptor Proteolysis : Implications for Platelet Function
Atherogenic lipid stress induces platelet hyperactivity through CD36-mediated hyposensitivity to prostacyclin: the role of phosphodiesterase 3A
Platelet factor 4-containing immune complexes induce platelet activation followed by calpain-dependent platelet death
Incorporation Of3H-Glycerol into Platelet Phospholipids During Platelet Aggregation
Genetic Loci Associated with Platelet Traits and Platelet Disorders
Platelet Factor XI
Platelet Contractile Proteins
Sticky Platelet Syndrome
What about Platelet Function in Platelet Concentrates?
Platelet immunology
Platelet Glycocalicin
Arachidonic acid metabolite 19(S)-HETE induces vasorelaxation and platelet inhibition by activating prostacyclin (IP) receptor
Platelet Membrane Receptor Proteolysis : Implications for Platelet Function
Atherogenic lipid stress induces platelet hyperactivity through CD36-mediated hyposensitivity to prostacyclin: the role of phosphodiesterase 3A
Platelet factor 4-containing immune complexes induce platelet activation followed by calpain-dependent platelet death
Incorporation Of3H-Glycerol into Platelet Phospholipids During Platelet Aggregation
Genetic Loci Associated with Platelet Traits and Platelet Disorders
Platelet Factor XI
Platelet Contractile Proteins
Sticky Platelet Syndrome
What about Platelet Function in Platelet Concentrates?
Platelet immunology
Platelet Glycocalicin
Arachidonic acid metabolite 19(S)-HETE induces vasorelaxation and platelet inhibition by activating prostacyclin (IP) receptor
Platelet Membrane Receptor Proteolysis : Implications for Platelet Function
Atherogenic lipid stress induces platelet hyperactivity through CD36-mediated hyposensitivity to prostacyclin: the role of phosphodiesterase 3A
Platelet factor 4-containing immune complexes induce platelet activation followed by calpain-dependent platelet death
Incorporation Of3H-Glycerol into Platelet Phospholipids During Platelet Aggregation
Genetic Loci Associated with Platelet Traits and Platelet Disorders
Platelet Factor XI
Platelet Contractile Proteins