Epigenetic regulation of spinal cord gene expression controls Opioid-Induced Hyperalgesia
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1744-8069
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Epigenetic regulation of spinal cord gene expression controls Opioid-Induced Hyperalgesia ; volume:10 ; number:1 ; day:12 ; month:9 ; year:2014 ; pages:1-12 ; date:12.2014
Molecular pain ; 10, Heft 1 (12.9.2014), 1-12, 12.2014
- Creator
-
Liang, De-Yong
Sun, Yuan
Shi, Xiao-You
Sahbaie, Peyman
Clark, J David
- Contributor
-
SpringerLink (Online service)
- DOI
-
10.1186/1744-8069-10-59
- URN
-
urn:nbn:de:101:1-2021081221263347578582
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
- 14.08.2025, 11:00 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Liang, De-Yong
- Sun, Yuan
- Shi, Xiao-You
- Sahbaie, Peyman
- Clark, J David
- SpringerLink (Online service)
Other Objects (12)
The multiple PDZ domain protein Mpdz/MUPP1 regulates opioid tolerance and opioid-induced hyperalgesia
FLT3 signaling inhibition abrogates opioid tolerance and hyperalgesia while preserving analgesia
Increased serum IL-6 level time-dependently regulates hyperalgesia and spinal mu opioid receptor expression during CFA-induced arthritis
Morphine-induced hyperalgesia involves mu opioid receptors and the metabolite morphine-3-glucuronide
Estrogen controls PKCepsilon-dependent mechanical hyperalgesia through direct action on nociceptive neurons
mTOR, a new potential target for chronic pain and opioid-induced tolerance and hyperalgesia
A Pharmacological Rationale to Reduce the Incidence of Opioid Induced Tolerance and Hyperalgesia: A Review
Increased spinal prodynorphin gene expression in reinflammation-associated hyperalgesia after neonatal inflammatory insult
Blocking spinal CCR2 with AZ889 reversed hyperalgesia in a model of neuropathic pain
OA08.03. Electroacupuncture alleviates hyperalgesia by inhibiting spinal interleukin-17 in an inflammatory pain rat model
Repeated electroacupuncture treatment attenuated hyperalgesia through suppression of spinal glial activation in chronic neuropathic pain rats
Activated spinal astrocytes are involved in the maintenance of chronic widespread mechanical hyperalgesia after cast immobilization
The multiple PDZ domain protein Mpdz/MUPP1 regulates opioid tolerance and opioid-induced hyperalgesia
FLT3 signaling inhibition abrogates opioid tolerance and hyperalgesia while preserving analgesia
Increased serum IL-6 level time-dependently regulates hyperalgesia and spinal mu opioid receptor expression during CFA-induced arthritis
Morphine-induced hyperalgesia involves mu opioid receptors and the metabolite morphine-3-glucuronide
Estrogen controls PKCepsilon-dependent mechanical hyperalgesia through direct action on nociceptive neurons
mTOR, a new potential target for chronic pain and opioid-induced tolerance and hyperalgesia
A Pharmacological Rationale to Reduce the Incidence of Opioid Induced Tolerance and Hyperalgesia: A Review
Increased spinal prodynorphin gene expression in reinflammation-associated hyperalgesia after neonatal inflammatory insult
Blocking spinal CCR2 with AZ889 reversed hyperalgesia in a model of neuropathic pain
OA08.03. Electroacupuncture alleviates hyperalgesia by inhibiting spinal interleukin-17 in an inflammatory pain rat model
Repeated electroacupuncture treatment attenuated hyperalgesia through suppression of spinal glial activation in chronic neuropathic pain rats
Activated spinal astrocytes are involved in the maintenance of chronic widespread mechanical hyperalgesia after cast immobilization
The multiple PDZ domain protein Mpdz/MUPP1 regulates opioid tolerance and opioid-induced hyperalgesia
FLT3 signaling inhibition abrogates opioid tolerance and hyperalgesia while preserving analgesia
Increased serum IL-6 level time-dependently regulates hyperalgesia and spinal mu opioid receptor expression during CFA-induced arthritis
Morphine-induced hyperalgesia involves mu opioid receptors and the metabolite morphine-3-glucuronide
Estrogen controls PKCepsilon-dependent mechanical hyperalgesia through direct action on nociceptive neurons
mTOR, a new potential target for chronic pain and opioid-induced tolerance and hyperalgesia
A Pharmacological Rationale to Reduce the Incidence of Opioid Induced Tolerance and Hyperalgesia: A Review
Increased spinal prodynorphin gene expression in reinflammation-associated hyperalgesia after neonatal inflammatory insult
Blocking spinal CCR2 with AZ889 reversed hyperalgesia in a model of neuropathic pain
OA08.03. Electroacupuncture alleviates hyperalgesia by inhibiting spinal interleukin-17 in an inflammatory pain rat model
Repeated electroacupuncture treatment attenuated hyperalgesia through suppression of spinal glial activation in chronic neuropathic pain rats