- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
1479-5876
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
Interleukin-2 alters distribution of CD144 (VE-cadherin) in endothelial cells ; volume:12 ; number:1 ; day:6 ; month:5 ; year:2014 ; pages:1-6 ; date:12.2014
Journal of translational medicine ; 12, Heft 1 (6.5.2014), 1-6, 12.2014
- Creator
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Kim, Dae Won
- Contributor
-
Zloza, Andrew
Broucek, Joseph
Schenkel, Jason M.
Ruby, Carl
Samaha, Georges
Kaufman, Howard L.
SpringerLink (Online service)
- DOI
-
10.1186/1479-5876-12-113
- URN
-
urn:nbn:de:1111-2016041448561
- Rights
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
11.08.2025, 8:59 PM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Kim, Dae Won
- Zloza, Andrew
- Broucek, Joseph
- Schenkel, Jason M.
- Ruby, Carl
- Samaha, Georges
- Kaufman, Howard L.
- SpringerLink (Online service)
Other Objects (12)
The relevance of VE-cadherin, ESAM and vinculin for endothelial junction integrity
The proteine tyrosine phosphatase VE-PTP controls endothelial cell contacts via VE-cadherin and Tie-2
ESAM controls opening of endothelial junctions via phosphorylation of VE-cadherin at Y685 residue
The role of VE-cadherin tyrosine phosphorylation for the control of endothelial cell contacts
Changes in expression of VE-cadherin and MMPs in endothelial cells: Implications for angiogenesis
Gene expression analysis of embryonic stem cells expressing VE-cadherin (CD144) during endothelial differentiation
Ubiquitin ligase CHFR mediated degradation of VE-cadherin through ubiquitylation disrupts endothelial adherens junctions
Interplay between neural-cadherin and vascular endothelial-cadherin in breast cancer progression
Vascular endothelial cadherin is an endostatin receptor
Interplay between N-cadherin and VE-cadherin in breast cancer progression
The C-terminus region of β-arrestin1 modulates VE-cadherin expression and endothelial cell permeability
ICAM‐1 Activates Platelets and Promotes Endothelial Permeability through VE‐Cadherin after Insufficient Radiofrequency Ablation
The relevance of VE-cadherin, ESAM and vinculin for endothelial junction integrity
The proteine tyrosine phosphatase VE-PTP controls endothelial cell contacts via VE-cadherin and Tie-2
ESAM controls opening of endothelial junctions via phosphorylation of VE-cadherin at Y685 residue
The role of VE-cadherin tyrosine phosphorylation for the control of endothelial cell contacts
Changes in expression of VE-cadherin and MMPs in endothelial cells: Implications for angiogenesis
Gene expression analysis of embryonic stem cells expressing VE-cadherin (CD144) during endothelial differentiation
Ubiquitin ligase CHFR mediated degradation of VE-cadherin through ubiquitylation disrupts endothelial adherens junctions
Interplay between neural-cadherin and vascular endothelial-cadherin in breast cancer progression
Vascular endothelial cadherin is an endostatin receptor
Interplay between N-cadherin and VE-cadherin in breast cancer progression
The C-terminus region of β-arrestin1 modulates VE-cadherin expression and endothelial cell permeability
ICAM‐1 Activates Platelets and Promotes Endothelial Permeability through VE‐Cadherin after Insufficient Radiofrequency Ablation
The relevance of VE-cadherin, ESAM and vinculin for endothelial junction integrity
The proteine tyrosine phosphatase VE-PTP controls endothelial cell contacts via VE-cadherin and Tie-2
ESAM controls opening of endothelial junctions via phosphorylation of VE-cadherin at Y685 residue
The role of VE-cadherin tyrosine phosphorylation for the control of endothelial cell contacts
Changes in expression of VE-cadherin and MMPs in endothelial cells: Implications for angiogenesis
Gene expression analysis of embryonic stem cells expressing VE-cadherin (CD144) during endothelial differentiation
Ubiquitin ligase CHFR mediated degradation of VE-cadherin through ubiquitylation disrupts endothelial adherens junctions
Interplay between neural-cadherin and vascular endothelial-cadherin in breast cancer progression
Vascular endothelial cadherin is an endostatin receptor
Interplay between N-cadherin and VE-cadherin in breast cancer progression
The C-terminus region of β-arrestin1 modulates VE-cadherin expression and endothelial cell permeability