LncRNA BLAT1 is Upregulated in Basal-like Breast Cancer through Epigenetic Modifications
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
2045-2322
- Extent
-
Online-Ressource
- Language
-
Englisch
- Notes
-
online resource.
- Bibliographic citation
-
LncRNA BLAT1 is Upregulated in Basal-like Breast Cancer through Epigenetic Modifications ; volume:8 ; number:1 ; day:22 ; month:10 ; year:2018 ; pages:1-11 ; date:12.2018
Scientific reports ; 8, Heft 1 (22.10.2018), 1-11, 12.2018
- Creator
-
Han, Yoo Jane
- Contributor
-
Boatman, Sonja M.
Zhang, Jing
Du, Xinxin C.
Yeh, Albert C.
Zheng, Yonglan
Mueller, Jeffrey
Olopade, Olufunmilayo I.
SpringerLink (Online service)
- DOI
-
10.1038/s41598-018-33629-y
- URN
-
urn:nbn:de:101:1-2018112122503395247066
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:51 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Han, Yoo Jane
- Boatman, Sonja M.
- Zhang, Jing
- Du, Xinxin C.
- Yeh, Albert C.
- Zheng, Yonglan
- Mueller, Jeffrey
- Olopade, Olufunmilayo I.
- SpringerLink (Online service)
Other Objects (12)
Feline mammary basal-like adenocarcinomas: a potential model for human triple-negative breast cancer (TNBC) with basal-like subtype
Inactivation of LATS1/2 drives luminal-basal plasticity to initiate basal-like mammary carcinomas
Weight loss reduces basal-like breast cancer through kinome reprogramming
Basal-like breast cancer: molecular profiles, clinical features and survival outcomes
Targeting BCL-2–expressing basal-like breast cancer with BH3-mimetics
The CD44+/CD24-phenotype is enriched in basal-like breast tumors
Activation of Bivalent Gene POU4F1 Promotes and Maintains Basal‐like Breast Cancer
Metabolic biomarkers for response to PI3K inhibition in basal-like breast cancer
Gene expression signatures of morphologically normal breast tissue identify basal-like tumors
ME1 promotes basal-like breast cancer progression and associates with poor prognosis
Increased gene expression variability in BRCA1-associated and basal-like breast tumours
MFAP5 promotes basal-like breast cancer progression by activating the EMT program
Feline mammary basal-like adenocarcinomas: a potential model for human triple-negative breast cancer (TNBC) with basal-like subtype
Inactivation of LATS1/2 drives luminal-basal plasticity to initiate basal-like mammary carcinomas
Weight loss reduces basal-like breast cancer through kinome reprogramming
Basal-like breast cancer: molecular profiles, clinical features and survival outcomes
Targeting BCL-2–expressing basal-like breast cancer with BH3-mimetics
The CD44+/CD24-phenotype is enriched in basal-like breast tumors
Activation of Bivalent Gene POU4F1 Promotes and Maintains Basal‐like Breast Cancer
Metabolic biomarkers for response to PI3K inhibition in basal-like breast cancer
Gene expression signatures of morphologically normal breast tissue identify basal-like tumors
ME1 promotes basal-like breast cancer progression and associates with poor prognosis
Increased gene expression variability in BRCA1-associated and basal-like breast tumours
MFAP5 promotes basal-like breast cancer progression by activating the EMT program
Feline mammary basal-like adenocarcinomas: a potential model for human triple-negative breast cancer (TNBC) with basal-like subtype
Inactivation of LATS1/2 drives luminal-basal plasticity to initiate basal-like mammary carcinomas
Weight loss reduces basal-like breast cancer through kinome reprogramming
Basal-like breast cancer: molecular profiles, clinical features and survival outcomes
Targeting BCL-2–expressing basal-like breast cancer with BH3-mimetics
The CD44+/CD24-phenotype is enriched in basal-like breast tumors
Activation of Bivalent Gene POU4F1 Promotes and Maintains Basal‐like Breast Cancer
Metabolic biomarkers for response to PI3K inhibition in basal-like breast cancer
Gene expression signatures of morphologically normal breast tissue identify basal-like tumors
ME1 promotes basal-like breast cancer progression and associates with poor prognosis
Increased gene expression variability in BRCA1-associated and basal-like breast tumours