NeuroD4 converts glioblastoma cells into neuron-like cells through the SLC7A11-GSH-GPX4 antioxidant axis
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
1 Online-Ressource.
- Language
-
Englisch
- Bibliographic citation
-
NeuroD4 converts glioblastoma cells into neuron-like cells through the SLC7A11-GSH-GPX4 antioxidant axis ; volume:9 ; number:1 ; day:15 ; month:8 ; year:2023 ; pages:1-12 ; date:12.2023
Cell death discovery ; 9, Heft 1 (15.8.2023), 1-12, 12.2023
- Creator
-
Wang, Hao
Zhao, Peiqi
Zhang, Ying
Chen, Zhen
Bao, Han
Qian, Wenqi
Wu, Jian
Xing, Zhenqiu
Hu, Xiaowei
Jin, Kunlin
Zhuge, Qichuan
Yang, Jianjing
- Contributor
-
SpringerLink (Online service)
- DOI
-
10.1038/s41420-023-01595-8
- URN
-
urn:nbn:de:101:1-2024021210163457044922
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
15.08.2025, 7:34 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Wang, Hao
- Zhao, Peiqi
- Zhang, Ying
- Chen, Zhen
- Bao, Han
- Qian, Wenqi
- Wu, Jian
- Xing, Zhenqiu
- Hu, Xiaowei
- Jin, Kunlin
- Zhuge, Qichuan
- Yang, Jianjing
- SpringerLink (Online service)
Other Objects (12)
Resveratrol reduces ROS-induced ferroptosis by activating SIRT3 and compensating the GSH/GPX4 pathway
Pien-Tze-Huang prevents hepatocellular carcinoma by inducing ferroptosis via inhibiting SLC7A11-GSH-GPX4 axis
Exogenous H2S initiating Nrf2/GPx4/GSH pathway through promoting Syvn1-Keap1 interaction in diabetic hearts
GIFT4 fusokine converts leukemic B cells into immune helper cells
Tumor-priming converts NK cells to memory-like NK cells
GSH 68er
The role and possible mechanism of the ferroptosis-related SLC7A11/GSH/GPX4 pathway in myocardial ischemia-reperfusion injury
Targeting redox homeostasis in rhabdomyosarcoma cells : GSH-depleting agents enhance auranofin-induced cell death
GPX4 overexpressed non-small cell lung cancer cells are sensitive to RSL3-induced ferroptosis
Gebläsehäcksler GSH 380
"B-RYAL" "GSh"
Gebläsesilohäcksler GSH 350
Resveratrol reduces ROS-induced ferroptosis by activating SIRT3 and compensating the GSH/GPX4 pathway
Pien-Tze-Huang prevents hepatocellular carcinoma by inducing ferroptosis via inhibiting SLC7A11-GSH-GPX4 axis
Exogenous H2S initiating Nrf2/GPx4/GSH pathway through promoting Syvn1-Keap1 interaction in diabetic hearts
GIFT4 fusokine converts leukemic B cells into immune helper cells
Tumor-priming converts NK cells to memory-like NK cells
GSH 68er
The role and possible mechanism of the ferroptosis-related SLC7A11/GSH/GPX4 pathway in myocardial ischemia-reperfusion injury
Targeting redox homeostasis in rhabdomyosarcoma cells : GSH-depleting agents enhance auranofin-induced cell death
GPX4 overexpressed non-small cell lung cancer cells are sensitive to RSL3-induced ferroptosis
Gebläsehäcksler GSH 380
"B-RYAL" "GSh"
Gebläsesilohäcksler GSH 350
Resveratrol reduces ROS-induced ferroptosis by activating SIRT3 and compensating the GSH/GPX4 pathway
Pien-Tze-Huang prevents hepatocellular carcinoma by inducing ferroptosis via inhibiting SLC7A11-GSH-GPX4 axis
Exogenous H2S initiating Nrf2/GPx4/GSH pathway through promoting Syvn1-Keap1 interaction in diabetic hearts
GIFT4 fusokine converts leukemic B cells into immune helper cells
Tumor-priming converts NK cells to memory-like NK cells
GSH 68er
The role and possible mechanism of the ferroptosis-related SLC7A11/GSH/GPX4 pathway in myocardial ischemia-reperfusion injury
Targeting redox homeostasis in rhabdomyosarcoma cells : GSH-depleting agents enhance auranofin-induced cell death
GPX4 overexpressed non-small cell lung cancer cells are sensitive to RSL3-induced ferroptosis
Gebläsehäcksler GSH 380
"B-RYAL" "GSh"