The differential contribution of tumour necrosis factor to thermal and mechanical hyperalgesia during chronic inflammation
- Standort
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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1478-6354
- Umfang
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Online-Ressource
- Sprache
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Englisch
- Anmerkungen
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online resource.
- Erschienen in
-
The differential contribution of tumour necrosis factor to thermal and mechanical hyperalgesia during chronic inflammation ; volume:7 ; number:4 ; day:12 ; month:4 ; year:2005 ; pages:1-10 ; date:6.2005
Arthritis Research & Therapy ; 7, Heft 4 (12.4.2005), 1-10, 6.2005
- Urheber
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Inglis, Julia J.
- Beteiligte Personen und Organisationen
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Nissim, Ahuva
Lees, Delphine M.
Hunt, Stephen P.
Chernajovsky, Yuti
Kidd, Bruce L.
SpringerLink (Online service)
- DOI
-
10.1186/ar1743
- URN
-
urn:nbn:de:1111-201605032756
- Rechteinformation
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
14.08.2025, 10:47 MESZ
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Beteiligte
- Inglis, Julia J.
- Nissim, Ahuva
- Lees, Delphine M.
- Hunt, Stephen P.
- Chernajovsky, Yuti
- Kidd, Bruce L.
- SpringerLink (Online service)
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Role of tumour necrosis factor alpha and monocyte subsets in vascular inflammation
Soluble epoxide hydrolase limits mechanical hyperalgesia during inflammation
Production of tumour necrosis factor during murine cutaneous leishmaniasis
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Linalyl Acetate Ameliorates Mechanical Hyperalgesia Through Suppressing Inflammation by TSLP/IL-33 Signaling
Polymorphism in the tumour necrosis factor receptor II gene is associated with circulating levels of soluble tumour necrosis factor receptors in rheumatoid arthritis
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Tumour necrosis factor-alpha expression in tumour islets confers a survival advantage in non-small cell lung cancer
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Role of tumour necrosis factor alpha and monocyte subsets in vascular inflammation
Soluble epoxide hydrolase limits mechanical hyperalgesia during inflammation
Production of tumour necrosis factor during murine cutaneous leishmaniasis
Clostridium Bacteria: Harnessing Tumour Necrosis for Targeted Gene Delivery
Linalyl Acetate Ameliorates Mechanical Hyperalgesia Through Suppressing Inflammation by TSLP/IL-33 Signaling
Polymorphism in the tumour necrosis factor receptor II gene is associated with circulating levels of soluble tumour necrosis factor receptors in rheumatoid arthritis
Exogenous tumour necrosis factor α induces suppression of autoimmune arthritis
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Tumour Necrosis Factor Alpha in Intestinal Homeostasis and Gut Related Diseases