DDIT3 Directs a Dual Mechanism to Balance Glycolysis and Oxidative Phosphorylation during Glutamine Deprivation
- Location
-
Deutsche Nationalbibliothek Frankfurt am Main
- Extent
-
Online-Ressource
- Language
-
Englisch
- Bibliographic citation
-
DDIT3 Directs a Dual Mechanism to Balance Glycolysis and Oxidative Phosphorylation during Glutamine Deprivation ; day:27 ; month:03 ; year:2021 ; extent:16
Advanced science ; (27.03.2021) (gesamt 16)
- Creator
-
Li, Mingyue
Thorne, Rick Francis
Shi, Ronghua
Zhang, Xu Dong
Li, Jingmin
Li, Jingtong
Zhang, Qingyuan
Wu, Mian
Liu, Lianxin
- DOI
-
10.1002/advs.202003732
- URN
-
urn:nbn:de:101:1-2021032815041594963501
- Rights
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
-
14.08.2025, 10:50 AM CEST
Data provider
This object is provided by:
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Li, Mingyue
- Thorne, Rick Francis
- Shi, Ronghua
- Zhang, Xu Dong
- Li, Jingmin
- Li, Jingtong
- Zhang, Qingyuan
- Wu, Mian
- Liu, Lianxin
Other Objects (12)
Glutamine deprivation counteracts hypoxia-induced chemoresistance
Glutamine synthetase is necessary for sarcoma adaptation to glutamine deprivation and tumor growth
Boosting Energy Deprivation by Synchronous Interventions of Glycolysis and Oxidative Phosphorylation for Bioenergetic Therapy Synergetic with Chemodynamic/Photothermal Therapy
PKM2 coordinates glycolysis with mitochondrial fusion and oxidative phosphorylation
Importance of glycolysis and oxidative phosphorylation in advanced melanoma
IKKβ activates p53 to promote cancer cell adaptation to glutamine deprivation
Glutamine deprivation alters the origin and function of cancer cell exosomes
HDAC8-dependent deacetylation of PKM2 directs nuclear localization and glycolysis to promote proliferation in hepatocellular carcinoma
PFKM inhibits doxorubicin-induced cardiotoxicity by enhancing oxidative phosphorylation and glycolysis
LAT2 regulates glutamine-dependent mTOR activation to promote glycolysis and chemoresistance in pancreatic cancer
Phosphorylation of PFKL regulates glycolysis in macrophages following pattern recognition receptor activation
Effects of glutamine deprivation on oxidative stress and cell survival in breast cell lines
Glutamine deprivation counteracts hypoxia-induced chemoresistance
Glutamine synthetase is necessary for sarcoma adaptation to glutamine deprivation and tumor growth
Boosting Energy Deprivation by Synchronous Interventions of Glycolysis and Oxidative Phosphorylation for Bioenergetic Therapy Synergetic with Chemodynamic/Photothermal Therapy
PKM2 coordinates glycolysis with mitochondrial fusion and oxidative phosphorylation
Importance of glycolysis and oxidative phosphorylation in advanced melanoma
IKKβ activates p53 to promote cancer cell adaptation to glutamine deprivation
Glutamine deprivation alters the origin and function of cancer cell exosomes
HDAC8-dependent deacetylation of PKM2 directs nuclear localization and glycolysis to promote proliferation in hepatocellular carcinoma
PFKM inhibits doxorubicin-induced cardiotoxicity by enhancing oxidative phosphorylation and glycolysis
LAT2 regulates glutamine-dependent mTOR activation to promote glycolysis and chemoresistance in pancreatic cancer
Phosphorylation of PFKL regulates glycolysis in macrophages following pattern recognition receptor activation
Effects of glutamine deprivation on oxidative stress and cell survival in breast cell lines
Glutamine deprivation counteracts hypoxia-induced chemoresistance
Glutamine synthetase is necessary for sarcoma adaptation to glutamine deprivation and tumor growth
Boosting Energy Deprivation by Synchronous Interventions of Glycolysis and Oxidative Phosphorylation for Bioenergetic Therapy Synergetic with Chemodynamic/Photothermal Therapy
PKM2 coordinates glycolysis with mitochondrial fusion and oxidative phosphorylation
Importance of glycolysis and oxidative phosphorylation in advanced melanoma
IKKβ activates p53 to promote cancer cell adaptation to glutamine deprivation
Glutamine deprivation alters the origin and function of cancer cell exosomes
HDAC8-dependent deacetylation of PKM2 directs nuclear localization and glycolysis to promote proliferation in hepatocellular carcinoma
PFKM inhibits doxorubicin-induced cardiotoxicity by enhancing oxidative phosphorylation and glycolysis
LAT2 regulates glutamine-dependent mTOR activation to promote glycolysis and chemoresistance in pancreatic cancer
Phosphorylation of PFKL regulates glycolysis in macrophages following pattern recognition receptor activation