A strong host response and lack of MYC expression are characteristic for diffuse large B cell lymphoma transformed from nodular lymphocyte predominant Hodgkin lymphoma
- Sprache
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Englisch
- Identifier
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1154589129
Thema
Krebs; Onkologie
Beteiligte Personen und Organisationen
Schuhmacher, Bianca
Rengstl, Benjamin
Döring, Claudia
Bein, Julia
Newrzela, Sebastian
Brunnberg, Uta
Kvasnicka, Hans Michael
Vornanen, Martine
Küppers, Ralf
Hansmann, Martin-Leo
Hartmann, Sylvia
Rengstl, Benjamin
Döring, Claudia
Bein, Julia
Newrzela, Sebastian
Brunnberg, Uta
Kvasnicka, Hans Michael
Vornanen, Martine
Küppers, Ralf
Hansmann, Martin-Leo
Hartmann, Sylvia
- URN
- Rechteinformation
-
Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
26.01.2023, 13:53 MEZ
Beteiligte
- Schuhmacher, Bianca
- Rengstl, Benjamin
- Döring, Claudia
- Bein, Julia
- Newrzela, Sebastian
- Brunnberg, Uta
- Kvasnicka, Hans Michael
- Vornanen, Martine
- Küppers, Ralf
- Hansmann, Martin-Leo
- Hartmann, Sylvia
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Diffuse large B cell lymphoma derived from nodular lymphocyte predominant Hodgkin lymphoma presents with variable histopathology
Nodular lymphocyte predominant hodgkin lymphoma and T cell/histiocyte rich large B cell lymphoma : endpoints of a spectrum of one disease? [[Elektronische Ressource]]
Migration properties distinguish tumor cells of classical hodgkin lymphoma from anaplastic large cell lymphoma cells
GLUT1 expression patterns in different Hodgkin lymphoma subtypes and progressively transformed germinal centers
Small and big Hodgkin-Reed-Sternberg cells of Hodgkin lymphoma cell lines L-428 and L-1236 lack consistent differences in gene expression profiles and are capable to reconstitute each other
Expression and functional relevance of cannabinoid receptor 1 in Hodgkin lymphoma
Ectopic expression of transcription factor BATF3 induces B-cell lymphomas in a murine B-cell transplantation model
Origin and pathogenesis of nodular lymphocyte–predominant Hodgkin lymphoma as revealed by global gene expression analysis
TNFAIP3 (A20) is a tumor suppressor gene in Hodgkin lymphoma and primary mediastinal B cell lymphoma
Basal autophagy is pivotal for Hodgkin and Reed-Sternberg cells' survival and growth revealing a new strategy for Hodgkin lymphoma treatment
Fibroblasts in nodular sclerosing classical hodgkin lymphoma are defined by a specific phenotype and protect tumor cells from brentuximab-vedotin induced injury
Lymphocyte predominant cells of nodular lymphocyte predominant Hodgkin lymphoma interact with rosetting T cells in an immunological synapse
Diffuse large B cell lymphoma derived from nodular lymphocyte predominant Hodgkin lymphoma presents with variable histopathology
Nodular lymphocyte predominant hodgkin lymphoma and T cell/histiocyte rich large B cell lymphoma : endpoints of a spectrum of one disease? [[Elektronische Ressource]]
Migration properties distinguish tumor cells of classical hodgkin lymphoma from anaplastic large cell lymphoma cells
GLUT1 expression patterns in different Hodgkin lymphoma subtypes and progressively transformed germinal centers
Small and big Hodgkin-Reed-Sternberg cells of Hodgkin lymphoma cell lines L-428 and L-1236 lack consistent differences in gene expression profiles and are capable to reconstitute each other
Expression and functional relevance of cannabinoid receptor 1 in Hodgkin lymphoma
Ectopic expression of transcription factor BATF3 induces B-cell lymphomas in a murine B-cell transplantation model
Origin and pathogenesis of nodular lymphocyte–predominant Hodgkin lymphoma as revealed by global gene expression analysis
TNFAIP3 (A20) is a tumor suppressor gene in Hodgkin lymphoma and primary mediastinal B cell lymphoma
Basal autophagy is pivotal for Hodgkin and Reed-Sternberg cells' survival and growth revealing a new strategy for Hodgkin lymphoma treatment
Fibroblasts in nodular sclerosing classical hodgkin lymphoma are defined by a specific phenotype and protect tumor cells from brentuximab-vedotin induced injury