TRPC Channels Activated by G Protein-Coupled Receptors Drive Ca2+ Dysregulation Leading to Secondary Brain Injury in the Mouse Model

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Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: Online-Ressource, 1 online resource.

Language: Englisch

Bibliographic citation: TRPC Channels Activated by G Protein-Coupled Receptors Drive Ca2+ Dysregulation Leading to Secondary Brain Injury in the Mouse Model ; day:18 ; month:7 ; year:2023 ; pages:1-15
Translational stroke research ; (18.7.2023), 1-15

Creator: Parmar, Jasneet
Jonquières, Georg von
Gorlamandala, Nagarajesh
Chung, Brandon
Craig, Amanda J.
Pinyon, Jeremy L.
Birnbaumer, Lutz
Klugmann, Matthias
Moorhouse, Andrew J.
Power, John M.
Housley, Gary D.

Contributor: SpringerLink (Online service)

DOI: 10.1007/s12975-023-01173-1

URN: urn:nbn:de:101:1-2023102412533617466050

Rights: Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 14.08.2025, 11:00 AM CEST

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Associated

Parmar, Jasneet
Jonquières, Georg von
Gorlamandala, Nagarajesh
Chung, Brandon
Craig, Amanda J.
Pinyon, Jeremy L.
Birnbaumer, Lutz
Klugmann, Matthias
Moorhouse, Andrew J.
Power, John M.
Housley, Gary D.
SpringerLink (Online service)

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TRPC Channels Activated by G Protein-Coupled Receptors Drive Ca2+ Dysregulation Leading to Secondary Brain Injury in the Mouse Model

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Other Objects (12)

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Chloroquine inhibits Ca2+ permeable ion channels-mediated Ca2+ signaling in primary B lymphocytes

Tetraspanin-13 modulates voltage-gated Cav2.2 Ca2+ channels

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PERK regulates Gq protein-coupled intracellular Ca2+ dynamics in primary cortical neurons

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Peripheral temperature dysregulation associated with functionally altered NaV1.8 channels

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