Aurora B‐dependent polarization of the cortical actomyosin network during mitotic exit
- Standort
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Deutsche Nationalbibliothek Frankfurt am Main
- Umfang
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Online-Ressource
- Sprache
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Englisch
- Erschienen in
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Aurora B‐dependent polarization of the cortical actomyosin network during mitotic exit ; volume:22 ; number:10 ; year:2021 ; extent:13
EMBO reports / European Molecular Biology Organization ; 22, Heft 10 (2021) (gesamt 13)
- Urheber
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Ramkumar, Nitya
Patel, Jigna V.
Anstatt, Jannis
Baum, Buzz
- DOI
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10.15252/embr.202152387
- URN
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urn:nbn:de:101:1-2021100515162360877622
- Rechteinformation
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
15.08.2025, 07:31 MESZ
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Beteiligte
- Ramkumar, Nitya
- Patel, Jigna V.
- Anstatt, Jannis
- Baum, Buzz
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Protein phosphatase 1 is essential for Greatwall inactivation at mitotic exit
CCAR2 controls mitotic progression through spatiotemporal regulation of Aurora B
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TRIP13 and APC15 drive mitotic exit by turnover of interphase- and unattached kinetochore-produced MCC
Recruitment of the mitotic exit network to yeast centrosomes couples septin displacement to actomyosin constriction
Aurora-A inactivation causes mitotic spindle pole fragmentation by unbalancing microtubule-generated forces
Mitotic polarization of transcription factors during asymmetric division establishes fate of forming cancer cells
Afadin controls cell polarization and mitotic spindle orientation in developing cortical radial glia
Phosphoproteome dynamics during mitotic exit in budding yeast
Mitotic drivers—inhibitors of the Aurora B Kinase
Candida albicans Cdc15 is essential for mitotic exit and cytokinesis
Protein phosphatase 1 is essential for Greatwall inactivation at mitotic exit
CCAR2 controls mitotic progression through spatiotemporal regulation of Aurora B
USP35 regulates mitotic progression by modulating the stability of Aurora B
The novel proteasomal substrate Far10 contributes to control of mitotic exit in yeast
TRIP13 and APC15 drive mitotic exit by turnover of interphase- and unattached kinetochore-produced MCC
Recruitment of the mitotic exit network to yeast centrosomes couples septin displacement to actomyosin constriction
Aurora-A inactivation causes mitotic spindle pole fragmentation by unbalancing microtubule-generated forces