Hochschulschrift
Cellular and molecular analysis of fracture healing in a neurofibromatosis type 1 conditional knockout mice model
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Dimensions
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30 cm
- Extent
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VI, 144 S.
- Language
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Englisch
- Notes
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Ill., graph. Darst.
Berlin, Humboldt-Univ., Diss., 2013
- Classification
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Medizin, Gesundheit
- Creator
- Table of contents
- Rights
-
Bei diesem Objekt liegt nur das Inhaltsverzeichnis digital vor. Der Zugriff darauf ist unbeschränkt möglich.
- Last update
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11.03.2025, 11:55 AM CET
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Object type
- Hochschulschrift
Associated
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Cellular and molecular analysis of fracture healing in a neurofibromatosis type 1 conditional knockout mice model
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Differences in fracture healing between female and male C57BL/6J mice
Role of midkine during fracture healing
Impaired Fracture Healing after Hemorrhagic Shock
The generation and characterisation of mice with conditional knock-out of the NMDAR subunit NR2B
Osteoblast-Specific Krm2 Overexpression and Lrp5 Deficiency Have Different Effects on Fracture Healing in Mice
Identification of the molecular role of Pelota protein (PELO) by analysis of conditional Pelo-knockout mice
Comparative analyses of Netherton syndrome patients and Spink5 conditional knock-out mice uncover disease-relevant pathways
Characterization of inducible SYK knockout mice
Cellular and molecular analysis of fracture healing in a neurofibromatosis type 1 conditional knockout mice model
Analysis of conditional Rer1 knockout mice
Characterization of pancreas-specific conditional Notch1 knockout mice
Oxygen treatment improves compromised fracture healing after thoracic trauma in mice
Differences in fracture healing between female and male C57BL/6J mice
Role of midkine during fracture healing
Impaired Fracture Healing after Hemorrhagic Shock
The generation and characterisation of mice with conditional knock-out of the NMDAR subunit NR2B
Osteoblast-Specific Krm2 Overexpression and Lrp5 Deficiency Have Different Effects on Fracture Healing in Mice
Identification of the molecular role of Pelota protein (PELO) by analysis of conditional Pelo-knockout mice
Comparative analyses of Netherton syndrome patients and Spink5 conditional knock-out mice uncover disease-relevant pathways