Iron commensalism of mesenchymal glioblastoma promotes ferroptosis susceptibility upon dopamine treatment
- Standort
-
Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
-
2399-3642
- Umfang
-
Online-Ressource
- Sprache
-
Englisch
- Anmerkungen
-
online resource.
- Erschienen in
-
Iron commensalism of mesenchymal glioblastoma promotes ferroptosis susceptibility upon dopamine treatment ; volume:5 ; number:1 ; day:16 ; month:6 ; year:2022 ; pages:1-14 ; date:12.2022
Communications biology ; 5, Heft 1 (16.6.2022), 1-14, 12.2022
- Urheber
-
Vo, Vu T. A.
Kim, Sohyun
Hua, Tuyen N. M.
Oh, Jiwoong
Jeong, Yangsik
- Beteiligte Personen und Organisationen
-
SpringerLink (Online service)
- DOI
-
10.1038/s42003-022-03538-y
- URN
-
urn:nbn:de:101:1-2022080623341824805935
- Rechteinformation
-
Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Letzte Aktualisierung
-
15.08.2025, 07:20 MESZ
Datenpartner
Dieses Objekt wird bereitgestellt von:
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Deutsche Nationalbibliothek. Bei Fragen zum Objekt wenden Sie sich bitte an den Datenpartner.
Beteiligte
- Vo, Vu T. A.
- Kim, Sohyun
- Hua, Tuyen N. M.
- Oh, Jiwoong
- Jeong, Yangsik
- SpringerLink (Online service)
Ähnliche Objekte (12)
Glioblastoma mesenchymal subtype enhances antioxidant defence to reduce susceptibility to ferroptosis
Inhibition of autophagy increases susceptibility of glioblastoma stem cells to temozolomide by igniting ferroptosis
ALDH1-mediated autophagy sensitizes glioblastoma cells to ferroptosis
Neutrophil-induced ferroptosis promotes tumor necrosis in glioblastoma progression
Pyroptosis, ferroptosis, and autophagy cross-talk in glioblastoma opens up new avenues for glioblastoma treatment
Proteogenomic characterization of ferroptosis regulators reveals therapeutic potential in glioblastoma
Oxidative degradation of dihydrofolate reductase increases CD38-mediated ferroptosis susceptibility
Increased susceptibility of cystic fibrosis airway epithelial cells to ferroptosis
RBMS1 interference inhibits malignant progression of glioblastoma cells and promotes ferroptosis
Enhanced sensitivity to ALDH1A3-dependent ferroptosis in TMZ-resistant glioblastoma cells
Exploring the Role of ALDH1A3 in RSL3-triggered Ferroptosis in Glioblastoma
Characterization of glioblastoma stem cells upon triggering of CD95
Glioblastoma mesenchymal subtype enhances antioxidant defence to reduce susceptibility to ferroptosis
Inhibition of autophagy increases susceptibility of glioblastoma stem cells to temozolomide by igniting ferroptosis
ALDH1-mediated autophagy sensitizes glioblastoma cells to ferroptosis
Neutrophil-induced ferroptosis promotes tumor necrosis in glioblastoma progression
Pyroptosis, ferroptosis, and autophagy cross-talk in glioblastoma opens up new avenues for glioblastoma treatment
Proteogenomic characterization of ferroptosis regulators reveals therapeutic potential in glioblastoma
Oxidative degradation of dihydrofolate reductase increases CD38-mediated ferroptosis susceptibility
Increased susceptibility of cystic fibrosis airway epithelial cells to ferroptosis
RBMS1 interference inhibits malignant progression of glioblastoma cells and promotes ferroptosis
Enhanced sensitivity to ALDH1A3-dependent ferroptosis in TMZ-resistant glioblastoma cells
Exploring the Role of ALDH1A3 in RSL3-triggered Ferroptosis in Glioblastoma
Characterization of glioblastoma stem cells upon triggering of CD95
Glioblastoma mesenchymal subtype enhances antioxidant defence to reduce susceptibility to ferroptosis
Inhibition of autophagy increases susceptibility of glioblastoma stem cells to temozolomide by igniting ferroptosis
ALDH1-mediated autophagy sensitizes glioblastoma cells to ferroptosis
Neutrophil-induced ferroptosis promotes tumor necrosis in glioblastoma progression
Pyroptosis, ferroptosis, and autophagy cross-talk in glioblastoma opens up new avenues for glioblastoma treatment
Proteogenomic characterization of ferroptosis regulators reveals therapeutic potential in glioblastoma
Oxidative degradation of dihydrofolate reductase increases CD38-mediated ferroptosis susceptibility
Increased susceptibility of cystic fibrosis airway epithelial cells to ferroptosis
RBMS1 interference inhibits malignant progression of glioblastoma cells and promotes ferroptosis
Enhanced sensitivity to ALDH1A3-dependent ferroptosis in TMZ-resistant glioblastoma cells
Exploring the Role of ALDH1A3 in RSL3-triggered Ferroptosis in Glioblastoma