Iron commensalism of mesenchymal glioblastoma promotes ferroptosis susceptibility upon dopamine treatment
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- ISSN
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2399-3642
- Extent
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Online-Ressource
- Language
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Englisch
- Notes
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online resource.
- Bibliographic citation
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Iron commensalism of mesenchymal glioblastoma promotes ferroptosis susceptibility upon dopamine treatment ; volume:5 ; number:1 ; day:16 ; month:6 ; year:2022 ; pages:1-14 ; date:12.2022
Communications biology ; 5, Heft 1 (16.6.2022), 1-14, 12.2022
- Creator
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Vo, Vu T. A.
Kim, Sohyun
Hua, Tuyen N. M.
Oh, Jiwoong
Jeong, Yangsik
- Contributor
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SpringerLink (Online service)
- DOI
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10.1038/s42003-022-03538-y
- URN
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urn:nbn:de:101:1-2022080623341824805935
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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15.08.2025, 7:20 AM CEST
Data provider
This object is provided by:
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Deutsche Nationalbibliothek. If you have any questions about the object, please contact the data provider.
Associated
- Vo, Vu T. A.
- Kim, Sohyun
- Hua, Tuyen N. M.
- Oh, Jiwoong
- Jeong, Yangsik
- SpringerLink (Online service)
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Proteogenomic characterization of ferroptosis regulators reveals therapeutic potential in glioblastoma
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Oxidative degradation of dihydrofolate reductase increases CD38-mediated ferroptosis susceptibility
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RBMS1 interference inhibits malignant progression of glioblastoma cells and promotes ferroptosis
Enhanced sensitivity to ALDH1A3-dependent ferroptosis in TMZ-resistant glioblastoma cells
Characterization of glioblastoma stem cells upon triggering of CD95
Glioblastoma mesenchymal subtype enhances antioxidant defence to reduce susceptibility to ferroptosis
Inhibition of autophagy increases susceptibility of glioblastoma stem cells to temozolomide by igniting ferroptosis
ALDH1-mediated autophagy sensitizes glioblastoma cells to ferroptosis
Neutrophil-induced ferroptosis promotes tumor necrosis in glioblastoma progression
Pyroptosis, ferroptosis, and autophagy cross-talk in glioblastoma opens up new avenues for glioblastoma treatment
Proteogenomic characterization of ferroptosis regulators reveals therapeutic potential in glioblastoma
Increased susceptibility of cystic fibrosis airway epithelial cells to ferroptosis
Oxidative degradation of dihydrofolate reductase increases CD38-mediated ferroptosis susceptibility
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RBMS1 interference inhibits malignant progression of glioblastoma cells and promotes ferroptosis
Enhanced sensitivity to ALDH1A3-dependent ferroptosis in TMZ-resistant glioblastoma cells