Inhibition of Cyclin D1 Expression in Human Glioblastoma Cells is Associated with Increased Temozolomide Chemosensitivity
- Location
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Deutsche Nationalbibliothek Frankfurt am Main
- Extent
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Online-Ressource
- Language
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Englisch
- Bibliographic citation
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Inhibition of Cyclin D1 Expression in Human Glioblastoma Cells is Associated with Increased Temozolomide Chemosensitivity ; volume:51 ; number:6 ; year:2019 ; pages:2496-2508
Cellular physiology and biochemistry ; 51, Heft 6 (2019), 2496-2508
- Creator
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Zhang, Danfeng
Dai, Dawei
Zhou, Mengxia
Li, Zhenxing
Wang, Chunhui
Lu, Yicheng
Li, Yiming
Wang, Junyu
- DOI
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10.1159/000495920
- URN
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urn:nbn:de:101:1-2019020109212599939545
- Rights
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Open Access; Der Zugriff auf das Objekt ist unbeschränkt möglich.
- Last update
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14.08.2025, 10:55 AM CEST
Data provider
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Associated
- Zhang, Danfeng
- Dai, Dawei
- Zhou, Mengxia
- Li, Zhenxing
- Wang, Chunhui
- Lu, Yicheng
- Li, Yiming
- Wang, Junyu
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Downregulating DNA methyltransferase 3B by suppressing the PI3K/Akt signaling pathway enhances the chemosensitivity of glioblastoma to temozolomide
Restoring chemo-sensitivity to temozolomide via targeted inhibition of poly (ADP-ribose) polymerase-1 by naringin in glioblastoma
Accelerated hyperfractionation plus temozolomide in glioblastoma
GINS2 regulates temozolomide chemosensitivity via the EGR1/ECT2 axis in gliomas
Knockdown ATG4C inhibits gliomas progression and promotes temozolomide chemosensitivity by suppressing autophagic flux
Temozolomide promotes genomic and phenotypic changes in glioblastoma cells
p53/E2F7 axis promotes temozolomide chemoresistance in glioblastoma multiforme
Considering the experimental use of temozolomide in glioblastoma research
Toxicity after radiochemotherapy for glioblastoma using temozolomide - a retrospective evaluation
Knockdown lncRNA CRNDE enhances temozolomide chemosensitivity by regulating autophagy in glioblastoma
Autophagy-based unconventional secretion of HMGB1 in glioblastoma promotes chemosensitivity to temozolomide through macrophage M1-like polarization
Long noncoding RNA AC003092.1 promotes temozolomide chemosensitivity through miR-195/TFPI-2 signaling modulation in glioblastoma
Downregulating DNA methyltransferase 3B by suppressing the PI3K/Akt signaling pathway enhances the chemosensitivity of glioblastoma to temozolomide
Restoring chemo-sensitivity to temozolomide via targeted inhibition of poly (ADP-ribose) polymerase-1 by naringin in glioblastoma
Accelerated hyperfractionation plus temozolomide in glioblastoma
GINS2 regulates temozolomide chemosensitivity via the EGR1/ECT2 axis in gliomas
Knockdown ATG4C inhibits gliomas progression and promotes temozolomide chemosensitivity by suppressing autophagic flux
Temozolomide promotes genomic and phenotypic changes in glioblastoma cells
p53/E2F7 axis promotes temozolomide chemoresistance in glioblastoma multiforme
Considering the experimental use of temozolomide in glioblastoma research
Toxicity after radiochemotherapy for glioblastoma using temozolomide - a retrospective evaluation
Knockdown lncRNA CRNDE enhances temozolomide chemosensitivity by regulating autophagy in glioblastoma
Autophagy-based unconventional secretion of HMGB1 in glioblastoma promotes chemosensitivity to temozolomide through macrophage M1-like polarization
Long noncoding RNA AC003092.1 promotes temozolomide chemosensitivity through miR-195/TFPI-2 signaling modulation in glioblastoma
Downregulating DNA methyltransferase 3B by suppressing the PI3K/Akt signaling pathway enhances the chemosensitivity of glioblastoma to temozolomide
Restoring chemo-sensitivity to temozolomide via targeted inhibition of poly (ADP-ribose) polymerase-1 by naringin in glioblastoma
Accelerated hyperfractionation plus temozolomide in glioblastoma
GINS2 regulates temozolomide chemosensitivity via the EGR1/ECT2 axis in gliomas
Knockdown ATG4C inhibits gliomas progression and promotes temozolomide chemosensitivity by suppressing autophagic flux
Temozolomide promotes genomic and phenotypic changes in glioblastoma cells
p53/E2F7 axis promotes temozolomide chemoresistance in glioblastoma multiforme
Considering the experimental use of temozolomide in glioblastoma research