P-TEFb Activation by RBM7 Shapes a Pro-survival Transcriptional Response to Genotoxic Stress

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Location: Deutsche Nationalbibliothek Frankfurt am Main

Extent: Online-Ressource

Language: Englisch

Bibliographic citation: In: Molecular Cell (2019) 74:254-267. https://doi.org/10.1016/j.molcel.2019.01.033

Event: Veröffentlichung

(where): Würzburg

(who): Universität Würzburg

(when): 2024

Creator: Bugai, Andrii
Quaresma, Alexandre J. C.
Friedel, Caroline C.
Lenasi, Tina
Düster, Robert
Sibley, Christopher R.
Fujinaga, Koh
Kukanja, Petra
Hennig, Thomas
Blasius, Melanie
Geyer, Matthias
Ule, Jernej
Dölken, Lars
Barborič, Matjaž

DOI: 10.1016/j.molcel.2019.01.033

URN: urn:nbn:de:bvb:20-opus-221726

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:36 AM CEST

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Associated

Bugai, Andrii
Quaresma, Alexandre J. C.
Friedel, Caroline C.
Lenasi, Tina
Düster, Robert
Sibley, Christopher R.
Fujinaga, Koh
Kukanja, Petra
Hennig, Thomas
Blasius, Melanie
Geyer, Matthias
Ule, Jernej
Dölken, Lars
Barborič, Matjaž
Universität Würzburg

Time of origin

2024

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P-TEFb Activation by RBM7 Shapes a Pro-survival Transcriptional Response to Genotoxic Stress

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Other Objects (12)

The Mycobacterium tuberculosis transcriptional landscape under genotoxic stress

Thresholds of Genotoxic and Non-Genotoxic Carcinogens

Telomerase gene expression bioassays indicate metabolic activation of genotoxic lower chlorinated polychlorinated biphenyls

Beta-catenin cleavage enhances transcriptional activation

Novel WYL domain-containing transcriptional activator acts in response to genotoxic stress in rapidly growing mycobacteria

Four functional genotoxic marker genes (Bax, Btg2, Ccng1, and Cdkn1a) discriminate genotoxic hepatocarcinogens from non-genotoxic hepatocarcinogens and non-genotoxic non-hepatocarcinogens in rat public toxicogenomics data, Open TG-GATEs

Programmable CRISPR‐Cas transcriptional activation in bacteria

Necdin modulates proliferative cell survival of human cells in response to radiation-induced genotoxic stress

Publisher Correction: Novel WYL domain-containing transcriptional activator acts in response to genotoxic stress in rapidly growing mycobacteria

Residual-QSAR. Implications for genotoxic carcinogenesis

Mutational signatures of genotoxic cancer therapies

Specific multivalent molecules boost CRISPR-mediated transcriptional activation

The Mycobacterium tuberculosis transcriptional landscape under genotoxic stress

Thresholds of Genotoxic and Non-Genotoxic Carcinogens

Telomerase gene expression bioassays indicate metabolic activation of genotoxic lower chlorinated polychlorinated biphenyls

Beta-catenin cleavage enhances transcriptional activation

Novel WYL domain-containing transcriptional activator acts in response to genotoxic stress in rapidly growing mycobacteria

Four functional genotoxic marker genes (Bax, Btg2, Ccng1, and Cdkn1a) discriminate genotoxic hepatocarcinogens from non-genotoxic hepatocarcinogens and non-genotoxic non-hepatocarcinogens in rat public toxicogenomics data, Open TG-GATEs

Programmable CRISPR‐Cas transcriptional activation in bacteria

Necdin modulates proliferative cell survival of human cells in response to radiation-induced genotoxic stress

Publisher Correction: Novel WYL domain-containing transcriptional activator acts in response to genotoxic stress in rapidly growing mycobacteria

Residual-QSAR. Implications for genotoxic carcinogenesis

Mutational signatures of genotoxic cancer therapies

Specific multivalent molecules boost CRISPR-mediated transcriptional activation

The Mycobacterium tuberculosis transcriptional landscape under genotoxic stress

Thresholds of Genotoxic and Non-Genotoxic Carcinogens

Telomerase gene expression bioassays indicate metabolic activation of genotoxic lower chlorinated polychlorinated biphenyls

Beta-catenin cleavage enhances transcriptional activation

Novel WYL domain-containing transcriptional activator acts in response to genotoxic stress in rapidly growing mycobacteria

Four functional genotoxic marker genes (Bax, Btg2, Ccng1, and Cdkn1a) discriminate genotoxic hepatocarcinogens from non-genotoxic hepatocarcinogens and non-genotoxic non-hepatocarcinogens in rat public toxicogenomics data, Open TG-GATEs

Programmable CRISPR‐Cas transcriptional activation in bacteria

Necdin modulates proliferative cell survival of human cells in response to radiation-induced genotoxic stress

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Residual-QSAR. Implications for genotoxic carcinogenesis

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