Mutations in dock1 disrupt early Schwann cell development

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Location: Deutsche Nationalbibliothek Frankfurt am Main

ISSN: 1749-8104

Extent: Online-Ressource

Language: Englisch

Notes: online resource.

Bibliographic citation: Mutations in dock1 disrupt early Schwann cell development ; volume:13 ; number:1 ; day:8 ; month:8 ; year:2018 ; pages:1-15 ; date:12.2018
Neural development ; 13, Heft 1 (8.8.2018), 1-15, 12.2018

Creator: Cunningham, Rebecca L.

Contributor: Herbert, Amy L.
Harty, Breanne L.
Ackerman, Sarah D.
Monk, Kelly R.
SpringerLink (Online service)

DOI: 10.1186/s13064-018-0114-9

URN: urn:nbn:de:101:1-2018091410550031394918

Rights: Der Zugriff auf das Objekt ist unbeschränkt möglich.

Last update: 15.08.2025, 7:23 AM CEST

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Cunningham, Rebecca L.
Herbert, Amy L.
Harty, Breanne L.
Ackerman, Sarah D.
Monk, Kelly R.
SpringerLink (Online service)

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Disease-associated mutations of claudin-19 disrupt retinal neurogenesis and visual function

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Extragenic suppressor mutations in ΔripA disrupt stability and function of LpxA

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De novo POGZ mutations in sporadic autism disrupt the DNA-binding activity of POGZ

Cancer-associated Histone H3 N-terminal arginine mutations disrupt PRC2 activity and impair differentiation

Two point mutations in protocadherin-1 disrupt hantavirus recognition and afford protection against lethal infection

Defects in hair cells disrupt the development of auditory peripheral circuitry

Mutations in the palm domain disrupt modulation of acid-sensing ion channel 1a currents by neuropeptides

Glucocerebrosidase mutations disrupt the lysosome and now the mitochondria

Affinity-optimizing enhancer variants disrupt development

Exon identity crisis: disease-causing mutations that disrupt the splicing code

Disease-associated mutations of claudin-19 disrupt retinal neurogenesis and visual function

APC mutations disrupt β-catenin destruction complex condensates organized by Axin phase separation

Extragenic suppressor mutations in ΔripA disrupt stability and function of LpxA

Maternal smoking and high BMI disrupt thyroid gland development

De novo POGZ mutations in sporadic autism disrupt the DNA-binding activity of POGZ

Cancer-associated Histone H3 N-terminal arginine mutations disrupt PRC2 activity and impair differentiation

Two point mutations in protocadherin-1 disrupt hantavirus recognition and afford protection against lethal infection

Defects in hair cells disrupt the development of auditory peripheral circuitry

Mutations in the palm domain disrupt modulation of acid-sensing ion channel 1a currents by neuropeptides

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Mutations in dock1 disrupt early Schwann cell development

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Other Objects (12)

Glucocerebrosidase mutations disrupt the lysosome and now the mitochondria

Affinity-optimizing enhancer variants disrupt development

Exon identity crisis: disease-causing mutations that disrupt the splicing code

Disease-associated mutations of claudin-19 disrupt retinal neurogenesis and visual function

APC mutations disrupt β-catenin destruction complex condensates organized by Axin phase separation

Extragenic suppressor mutations in ΔripA disrupt stability and function of LpxA

Maternal smoking and high BMI disrupt thyroid gland development

De novo POGZ mutations in sporadic autism disrupt the DNA-binding activity of POGZ

Cancer-associated Histone H3 N-terminal arginine mutations disrupt PRC2 activity and impair differentiation

Two point mutations in protocadherin-1 disrupt hantavirus recognition and afford protection against lethal infection

Defects in hair cells disrupt the development of auditory peripheral circuitry

Mutations in the palm domain disrupt modulation of acid-sensing ion channel 1a currents by neuropeptides

Glucocerebrosidase mutations disrupt the lysosome and now the mitochondria

Affinity-optimizing enhancer variants disrupt development

Exon identity crisis: disease-causing mutations that disrupt the splicing code

Disease-associated mutations of claudin-19 disrupt retinal neurogenesis and visual function

APC mutations disrupt β-catenin destruction complex condensates organized by Axin phase separation

Extragenic suppressor mutations in ΔripA disrupt stability and function of LpxA

Maternal smoking and high BMI disrupt thyroid gland development

De novo POGZ mutations in sporadic autism disrupt the DNA-binding activity of POGZ

Cancer-associated Histone H3 N-terminal arginine mutations disrupt PRC2 activity and impair differentiation

Two point mutations in protocadherin-1 disrupt hantavirus recognition and afford protection against lethal infection

Defects in hair cells disrupt the development of auditory peripheral circuitry

Mutations in the palm domain disrupt modulation of acid-sensing ion channel 1a currents by neuropeptides

Glucocerebrosidase mutations disrupt the lysosome and now the mitochondria

Affinity-optimizing enhancer variants disrupt development

Exon identity crisis: disease-causing mutations that disrupt the splicing code

Disease-associated mutations of claudin-19 disrupt retinal neurogenesis and visual function

APC mutations disrupt β-catenin destruction complex condensates organized by Axin phase separation

Extragenic suppressor mutations in ΔripA disrupt stability and function of LpxA

Maternal smoking and high BMI disrupt thyroid gland development

De novo POGZ mutations in sporadic autism disrupt the DNA-binding activity of POGZ

Cancer-associated Histone H3 N-terminal arginine mutations disrupt PRC2 activity and impair differentiation

Two point mutations in protocadherin-1 disrupt hantavirus recognition and afford protection against lethal infection

Defects in hair cells disrupt the development of auditory peripheral circuitry

Mutations in the palm domain disrupt modulation of acid-sensing ion channel 1a currents by neuropeptides

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